Nesheim B I, Saetre T
Maturitas. 1982 Apr;4(1):49-55. doi: 10.1016/0378-5122(82)90019-6.
Blood velocity changes in the radial, temporal and lateral thoracic arteries have been measured quantitatively during climacteric hot flushes by means of an ultrasonic velocity meter. In the radial artery the median increase in velocity was 6 times greater than the basal velocity (range 4-10); in both the temporal and the lateral thoracic arteries the median increase was 1.5 (1.5-3). Simultaneous temperature measurements showed a fairly high increase in finger temperatures. An initial increase in temperature on the forehead was followed by a decrease. The core temperature measured on the tympanic membrane decreased. The pattern of blood velocity which increases during hot flushes, is identical to the pattern seen when someone is transferred to an uncomfortably high temperature. This similarity points to the hot flush as a time in which a heat loss occurs. As temperature measurements show no accumulation of heat prior to the flushes, it is reasonable to assume that hot flushes are the result of a central disorder of temperature regulation.
在更年期潮热期间，通过超声速度仪对桡动脉、颞动脉和胸外侧动脉的血流速度变化进行了定量测量。在桡动脉中，血流速度的中位数增加是基础速度的6倍（范围为4至10）；在颞动脉和胸外侧动脉中，中位数增加均为1.5（1.5至3）。同时进行的温度测量显示手指温度有相当高的升高。前额温度先升高，随后下降。鼓膜测量的核心温度降低。潮热期间血流速度增加的模式与将某人转移到不适宜的高温环境时观察到的模式相同。这种相似性表明潮热是发生热量散失的时期。由于温度测量显示在潮热之前没有热量积累，因此合理的假设是潮热是体温调节中枢紊乱的结果。
