Nicassio P M, Boylan M B, McCabe T G
Br J Med Psychol. 1982 Jun;55(Pt 2):159-66. doi: 10.1111/j.2044-8341.1982.tb01494.x.
This paper describes a study in which progressive relaxation, EMG frontalis biofeedback, and a biofeedback placebo manipulation were compared in the treatment of severe insomnia with 40 chronically sleep-disturbed adult patients. Progressive relaxation and EMG biofeedback led to significant reductions in both reported sleep-onset latency and depressive symptomatology. However, when compared individually with the biofeedback placebo group, neither progressive relaxation nor EMG biofeedback emerged as significantly more effective on the sleep-onset latency measure. The clinical improvement in the biofeedback placebo group suggests that expectancies related to the ability to relax may contribute to the therapeutic effectiveness of relaxation strategies, even with chronic, severely disturbed insomniacs. Improvement in sleep-onset latency was not significantly related to reductions in frontalis EMG activity either within or between groups, a finding which raises questions concerning the clinical role and importance of physiological relaxation in the treatment of sleep-onset disturbance.
本文描述了一项研究,该研究对40名长期睡眠障碍的成年严重失眠患者采用渐进性放松、额肌肌电图生物反馈以及生物反馈安慰剂操作进行治疗,并对三者进行了比较。渐进性放松和肌电图生物反馈均使报告的入睡潜伏期和抑郁症状显著减轻。然而,当与生物反馈安慰剂组单独比较时,无论是渐进性放松还是肌电图生物反馈在入睡潜伏期测量上都未显示出显著更有效。生物反馈安慰剂组的临床改善表明,即使对于慢性、严重睡眠障碍的失眠症患者,与放松能力相关的期望可能有助于放松策略的治疗效果。在组内或组间,入睡潜伏期的改善与额肌肌电图活动的降低均无显著相关性,这一发现引发了关于生理放松在治疗入睡障碍中的临床作用和重要性的疑问。