• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利:CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法,用中文像聊天一样搜索,搜遍4000万医学文献。AI智能推荐,让科研检索更轻松。

立即免费搜索

文件翻译

保留排版,准确专业,支持PDF/Word/PPT等文件格式,支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述,25分钟生成高质量综述,智能提取关键信息,辅助科研写作。

立即免费体验

刚果锥虫感染犊牛的发病机制。I. 临床观察与大体病理变化

The pathogenesis of Trypanosoma congolense infection in calves. I. Clinical observations and gross pathological changes.

作者信息

Valli V E, Forsberg C M, Robinson G A

出版信息

Vet Pathol. 1978 Sep;15(5):608-20. doi: 10.1177/030098587801500504.

DOI:10.1177/030098587801500504
PMID:716157
Abstract

Holstein calves infected with Trypanosoma congolense TREU 112 had intermittent fever, debility and a poor hair coat. At postmortem examination the most significant findings were enlargement of the liver, kidneys and spleen and increased volume of hematopoietic marrow. A group of 13 calves infected for 18 weeks had marked enlargement of lymph nodes throughout the body and hemal nodes were prominent in the sublumbar area caudal to the kidneys. Another group infected for 7 weeks had no increase in the weight of lymph nodes. Studies with 51Cr labelled autologous red cells given shortly before postmortem examination showed that the liver was the most active organ with respect to sequestration of labelled red cells.

摘要

感染刚果锥虫TREU 112的荷斯坦犊牛出现间歇性发热、虚弱和被毛状况不佳。尸检时最显著的发现是肝脏、肾脏和脾脏肿大以及造血骨髓体积增加。一组13头感染18周的犊牛全身淋巴结明显肿大,肾尾侧腰下区域的血淋巴结突出。另一组感染7周的犊牛淋巴结重量未增加。在尸检前不久给予51Cr标记的自体红细胞进行的研究表明,就标记红细胞的滞留而言,肝脏是最活跃的器官。

相似文献

1
The pathogenesis of Trypanosoma congolense infection in calves. I. Clinical observations and gross pathological changes.刚果锥虫感染犊牛的发病机制。I. 临床观察与大体病理变化
Vet Pathol. 1978 Sep;15(5):608-20. doi: 10.1177/030098587801500504.
2
The pathogenesis of Trypanosoma congolense infection in calves. V. Quantitative histological changes.刚果锥虫感染犊牛的发病机制。V. 组织学定量变化
Vet Pathol. 1979 May;16(3):334-68. doi: 10.1177/030098587901600307.
3
The pathogenesis of Trypanosoma congolense infection in calves. II. Anemia and erythroid response.刚果锥虫感染犊牛的发病机制。II. 贫血与红系反应。
Vet Pathol. 1978 Nov;15(6):732-45. doi: 10.1177/030098587801500605.
4
The pathogenesis of infection with Trypanosoma vivax in goats and cattle.
Vet Rec. 1980 Dec 13;107(24):551-7. doi: 10.1136/vr.107.24.551.
5
Trypanosoma congolense. II. Histopathologic findings in experimentally infected cattle.刚果锥虫。二、实验感染牛的组织病理学发现。
Exp Parasitol. 1974 Aug;36(1):20-6. doi: 10.1016/0014-4894(74)90108-8.
6
Trypanosomes in the lymph nodes of cattle and sheep infected with Trypanosoma congolense.
Res Vet Sci. 1979 Jul;27(1):129-31.
7
The quantitation of Trypanosoma congolense in calves. Iv. In vitro culture of myeloid and erythroid marrow cells.刚果锥虫在犊牛体内的定量研究。四、髓系和红系骨髓细胞的体外培养
Tropenmed Parasitol. 1980 Dec;31(4):425-34.
8
A comparative study of the responses of the thymus, spleen, lymph nodes and bone marrow of the albino rat to infection with Trypanosoma congolense and Trypanosoma brucei.对白化大鼠的胸腺、脾脏、淋巴结和骨髓对刚果锥虫和布氏锥虫感染反应的比较研究。
Res Vet Sci. 1977 Sep;23(2):196-203.
9
The pathogenesis of Trypanosoma congolense infection in calves. III. Neutropenia and myeloid response.刚果锥虫感染犊牛的发病机制。III. 中性粒细胞减少和髓系反应。
Vet Pathol. 1979 Jan;16(1):96-107. doi: 10.1177/030098587901600110.
10
The pathology of Trypanosoma brucei infection in the rat.
Res Vet Sci. 1974 Jan;16(1):77-84.

引用本文的文献

1
Identifying Cattle Breed-Specific Partner Choice of Transcription Factors during the African Trypanosomiasis Disease Progression Using Bioinformatics Analysis.利用生物信息学分析确定非洲锥虫病疾病进展过程中转录因子的牛品种特异性伴侣选择
Vaccines (Basel). 2020 May 23;8(2):246. doi: 10.3390/vaccines8020246.
2
Tissue tropism in parasitic diseases.寄生虫病的组织嗜性。
Open Biol. 2019 May 31;9(5):190036. doi: 10.1098/rsob.190036.
3
Species-Specific Adaptations of Trypanosome Morphology and Motility to the Mammalian Host.锥虫形态和运动对哺乳动物宿主的物种特异性适应
PLoS Pathog. 2016 Feb 12;12(2):e1005448. doi: 10.1371/journal.ppat.1005448. eCollection 2016 Feb.