Wallin B G, Nerhed C
J Auton Nerv Syst. 1982 Nov;6(3):293-302. doi: 10.1016/0165-1838(82)90002-9.
Pulse-synchronous bursts of sympathetic vasoconstrictor impulses in peroneal muscle nerve fascicles were recorded together with intra-arterial blood pressure and ECG in 24 healthy subjects resting in the supine position. A statistical procedure similar to spike-triggered averaging was used to calculate mean changes of blood pressure and pulse interval following a burst of sympathetic impulses. In all subjects the result was a transient increase of blood pressure and a transient shortening of pulse interval with maxima occurring on the average 5.5 and 2.9s, respectively, after the burst. It is suggested that the same baroreceptor events that relate to a burst of sympathetic impulses in muscle nerves also change cardiac autonomic outflow to cause transient cardioacceleration. Presumably, the transient blood pressure increase following the burst is due to the combined effects of cardioacceleration and peripheral vasoconstriction.
在24名仰卧位休息的健康受试者中，记录了腓骨肌神经束中与动脉内血压和心电图同步的交感缩血管冲动的脉冲。采用一种类似于脉冲触发平均法的统计程序，来计算交感神经冲动爆发后血压和脉搏间期的平均变化。在所有受试者中，结果都是血压短暂升高，脉搏间期短暂缩短，最大值分别平均出现在冲动爆发后的5.5秒和2.9秒。有人提出，与肌肉神经中交感神经冲动爆发相关的相同压力感受器事件，也会改变心脏自主神经输出，从而导致短暂的心动加速。据推测，冲动爆发后血压的短暂升高是心动加速和外周血管收缩共同作用的结果。
