Delise P, D'Este D, Monico E, Millosevich P, Raviele A, Dainese F, Piccolo E
G Ital Cardiol. 1980;10(12):1588-93.
Three cases of accelerated idioventricular rhythm with isorhythmic A-V dissociation are presented. One case was affected by hypertrophic non obstructive myocardiopathy and two cases were affected by coronary heart disease with previous myocardial infarction. In all the cases of the arrhythmia persisted for days or months. By means of the electrophysiological study we could establish that 1) the focus was not protected by entrance block and 2) it was localized in the main branches of the conduction system. In the first case the focus was localized in the right bundle branch, in the second case, it was localized in the anterior fascicle of the left bundle branch and in the third case in the posterior fascicle of the left bundle branch. These electrophysiological characteristics suggest that the arrhythmia was due to increased firing of normal idioventricular pacemakers. On the basis of this electrogenetic interpretation we outline that in our cases the term of accelerated idioventricular rhythm should be preferred to others used in literature such as slow ventricular tachycardia.
本文报告3例伴有等律性房室分离的加速性心室自主节律。1例患有肥厚性非梗阻性心肌病,2例患有冠心病并曾有过心肌梗死。所有病例中,心律失常均持续数天或数月。通过电生理研究,我们可以确定:1)起搏点未受传入阻滞保护;2)起搏点位于传导系统的主要分支。第1例中,起搏点位于右束支;第2例中,起搏点位于左束支前分支;第3例中,起搏点位于左束支后分支。这些电生理特征提示,心律失常是由于正常心室自主起搏点的发放增加所致。基于这种电发生学解释,我们概述,在我们的病例中,加速性心室自主节律这一术语比文献中使用的其他术语(如缓慢型室性心动过速)更可取。