内毒素对荷瘤小鼠和对细菌病原体有反应的小鼠的毒性增加：巨噬细胞激活作为一个共同因素。
Increased toxicity of endotoxin for tumor-bearing mice and mice responding to bacterial pathogens: macrophage activation as a common denominator.
作者信息
Berendt M J, Newborg M F, North R J
出版信息
Infect Immun. 1980 May;28(2):645-7. doi: 10.1128/iai.28.2.645-647.1980.
Abstract
Mice bearing the syngeneic SA-1 sarcoma or treated with live Mycobacterium bovis BCG or Formalin-killed Corynebacterium parvum acquired a greatly increased susceptibility to the lethal effects of endotoxin. In all three experimental models, the acquisition of increased sensitivity to endotoxin was concordant with the generation of a systemically activated macrophage system.
摘要
携带同基因SA-1肉瘤的小鼠，或用活的牛型结核分枝杆菌卡介苗或福尔马林灭活的短小棒状杆菌处理过的小鼠，对内毒素的致死作用敏感性大大增加。在所有这三种实验模型中，对内毒素敏感性的增加与全身活化的巨噬细胞系统的产生是一致的。
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