Seidman D S, Shiloh M, Stevenson D K, Vreman H J, Gale R
Department of Neonatology, Bikur-Cholim Hospital, Jerusalem, Israel.
J Pediatr. 1995 Nov;127(5):804-6. doi: 10.1016/s0022-3476(95)70177-x.
End-tidal carbon monoxide was measured in 108 newborn infants who had been screened for glucose-6-phosphate dehydrogenase (G6PD) deficiency. The mean +/- SD end-tidal carbon monoxide did not differ significantly between the G6PD-deficient and the normal neonates, 2.1 +/- 0.6 microliters/L and 2.0 +/- 0.5 microliters/L, respectively, within 12 hours of birth and 1.9 +/- 1.4 microliters/L and 1.5 +/- 0.7 microliters/L, respectively, at 48 to 72 hours after birth. On the basis of these measurements, hemolysis is not a sufficient explanation for jaundice in G6PD-deficient newborn infants in the transitional period.
对108名曾接受葡萄糖-6-磷酸脱氢酶（G6PD）缺乏症筛查的新生儿进行了潮气末一氧化碳测定。在出生后12小时内，G6PD缺乏的新生儿和正常新生儿的平均±标准差潮气末一氧化碳水平无显著差异，分别为2.1±0.6微升/升和2.0±0.5微升/升；在出生后48至72小时，分别为1.9±1.4微升/升和1.5±0.7微升/升。基于这些测量结果，溶血不足以解释过渡期G6PD缺乏的新生儿黄疸。
