Ul'yaninskii L S
Laboratory of Experimental Cardiology, P.K. Anokhin Scientific Research Institute of Normal Physiology, Moscow.
Neurosci Behav Physiol. 1995 May-Jun;25(3):257-65. doi: 10.1007/BF02360215.
It has been demonstrated in chronic experiments on animals (rabbits, rats) that in the presence of a predominance of sympathetic influences on the heart during emotional stress, the disturbances that appear in metabolism and myocardial ultrastructure promote a decrease in the electrical stability of the heart, the development of disturbances in cardiac rhythm, and arterial hypertension. A predominance of parasympathetic influences on the heart during stress, to the contrary, leads to an increase in the electrical stability of the heart, and also impedes an increase in the content of catecholamines and the development of structural injuries in the myocardium. At the same time, arterial hypertension is absent. An inference is drawn regarding the adaptive-trophic, protective action of the vagus nerves on the heart during emotional stress.
在对动物（兔子、大鼠）进行的慢性实验中已证明，在情绪应激期间交感神经对心脏的影响占主导时，代谢和心肌超微结构出现的紊乱会促使心脏电稳定性降低、心律紊乱的发生以及动脉高血压。相反，在应激期间副交感神经对心脏的影响占主导时，会导致心脏电稳定性增加，并且还会阻碍儿茶酚胺含量的增加以及心肌结构损伤的发展。同时，不会出现动脉高血压。由此推断出迷走神经在情绪应激期间对心脏具有适应性营养保护作用。
