Ul'ianinskiĭ L S
Fiziol Zh Im I M Sechenova. 1994 Feb;80(2):23-33.
Chronic experiments in rabbits and rats revealed that, when sympathetic influences on the heart prevail during emotional stress, reduce the heart electric stability, induce disorders in the heart rhythm and lead to arterial hypertension. Prevailing of parasympathetic influences, on the contrary, augments the heart electric stability, interferes with an increase in the catecholamines content and prevents structural lesions in the myocardium. Arterial hypertension is absent, at that. The vagal nerves seem to exert an adaptational-trophic and defensive effects on the heart in emotional stress.
对兔子和大鼠进行的慢性实验表明，在情绪应激期间，当交感神经对心脏的影响占主导时，会降低心脏电稳定性，诱发心律紊乱并导致动脉高血压。相反，副交感神经影响占主导时，则会增强心脏电稳定性，抑制儿茶酚胺含量增加并防止心肌出现结构性损伤。此时不会出现动脉高血压。迷走神经似乎在情绪应激中对心脏发挥适应性营养和防御作用。
