Ul'ianinskiĭ L S, Kosharskaia I L, Starinskiĭ Iu G, Beskrovnova N N, Bogdanovich N K
Kardiologiia. 1986 May;26(5):71-5.
The development of ventricular fibrillation associated with experimental emotional stress was studied in chronic rabbit experiments. Changes in myocardial-fibre action potentials, thresholds for ventricular fibrillation and its precursors, blood and myocardial catecholamine levels and heart-muscle ultrastructure were examined. Emotional stress was shown to increase, via sympathoadrenal activation, functional and structural heterogeneity of the myocardium, leading, alongside other causes, to arrhythmias and death from ventricular fibrillation. Moderately prevailing parasympathetic effects on the heart during stress exposure contribute to the elevation of fibrillation and fibrillation precursor thresholds and prevent catecholamine rise and myocardial structural damage.
在慢性兔实验中研究了与实验性情绪应激相关的心室颤动的发展。检测了心肌纤维动作电位、心室颤动及其先兆的阈值、血液和心肌儿茶酚胺水平以及心肌超微结构的变化。结果表明，情绪应激通过交感-肾上腺激活增加心肌的功能和结构异质性，与其他原因一起导致心律失常和心室颤动死亡。在应激暴露期间适度占优势的副交感神经对心脏的作用有助于提高颤动和颤动先兆阈值，并防止儿茶酚胺升高和心肌结构损伤。
