Engelberts I, Tordoir J H, Boon E S, Schreij G
Department of Surgery, Academic Hospital, Maastricht, The Netherlands.
Am J Nephrol. 1995;15(4):323-6. doi: 10.1159/000168857.
A dialysis arteriovenous fistula caused life-threatening high-output cardiac failure in a 66-year-old patient. Excessive shunting through the dialysis fistula was demonstrated by invasive measurement of cardiac output, systemic arterial blood pressure, systemic vascular resistance, and oxygen consumption before and after temporary occlusion of the dialysis fistula. Noninvasive echocardiographic evaluation of the influence of fistula compression on cardiac output and noninvasive duplex measurement of the fistula flow also confirmed the diagnosis. Following surgical closure of the fistula, the patient's condition improved, and signs of congestive heart failure subsided. High-output cardiac failure is a rare complication of dialysis arteriovenous fistulas. The diagnosis may remain unrecognized for longer periods. Noninvasive methods for estimation of the hemodynamic importance of a fistula may be of help in the establishment of the proper diagnosis.
一名66岁患者因透析动静脉内瘘引发了危及生命的高输出量心力衰竭。通过在临时阻断透析内瘘前后对心输出量、体动脉血压、体循环血管阻力和氧耗进行有创测量,证实了经透析内瘘的分流过多。对瘘管压迫对心输出量影响的无创超声心动图评估以及对瘘管血流量的无创双功测量也证实了该诊断。在手术闭合内瘘后,患者病情改善,充血性心力衰竭体征消退。高输出量心力衰竭是透析动静脉内瘘的一种罕见并发症。该诊断可能在较长时间内未被识别。估计内瘘血流动力学重要性的无创方法可能有助于做出正确诊断。