Animal models in ANCA-vasculitis.
作者信息
Kettritz R, Yang J J, Kinjoh K, Jennette J C, Falk R J
机构信息
Department of Medicine and Pathology, University of North Carolina at Chapel Hill, USA.
出版信息
Clin Exp Immunol. 1995 Jul;101 Suppl 1(Suppl 1):12-5. doi: 10.1111/j.1365-2249.1995.tb06154.x.
Abstract
摘要
相似文献
1
Animal models in ANCA-vasculitis.抗中性粒细胞胞浆抗体相关性血管炎的动物模型
Clin Exp Immunol. 1995 Jul;101 Suppl 1(Suppl 1):12-5. doi: 10.1111/j.1365-2249.1995.tb06154.x.
2
Animal models of systemic vasculitis.系统性血管炎的动物模型
J Autoimmun. 1993 Apr;6(2):251-64. doi: 10.1006/jaut.1993.1022.
3
Pathogenesis of lupus nephritis and ANCA-associated systemic vasculitis: lessons from experimental models.
Am J Kidney Dis. 1998 May;31(5):I-Iii.
4
5
Anti-oxidized low-density lipoprotein antibodies in myeloperoxidase-positive vasculitis patients preferentially recognize hypochlorite-modified low density lipoproteins.髓过氧化物酶阳性血管炎患者中的抗氧化低密度脂蛋白抗体优先识别次氯酸盐修饰的低密度脂蛋白。
Clin Exp Immunol. 2007 Aug;149(2):257-64. doi: 10.1111/j.1365-2249.2007.03420.x. Epub 2007 May 22.
6
Bone marrow-derived cells are sufficient and necessary targets to mediate glomerulonephritis and vasculitis induced by anti-myeloperoxidase antibodies.骨髓来源的细胞是介导抗髓过氧化物酶抗体诱导的肾小球肾炎和血管炎的充分必要靶点。
J Am Soc Nephrol. 2006 Dec;17(12):3355-64. doi: 10.1681/ASN.2006070718. Epub 2006 Nov 15.
7
Pathophysiology of ANCA-associated vasculitides: are ANCA really pathogenic?抗中性粒细胞胞浆抗体相关血管炎的病理生理学：抗中性粒细胞胞浆抗体真的具有致病性吗？
Kidney Int. 2004 May;65(5):1564-7. doi: 10.1111/j.1523-1755.2004.05412.x.
8
9
L8. Animal models of ANCA associated vasculitis: the contribution of autoantibodies and autoreactive T cells.L8. 抗中性粒细胞胞浆抗体相关血管炎的动物模型：自身抗体和自身反应性T细胞的作用
Presse Med. 2013 Apr;42(4 Pt 2):515-7. doi: 10.1016/j.lpm.2013.01.008. Epub 2013 Feb 28.
10
Antineutrophil cytoplasmic antibody (ANCA)-associated autoimmune diseases induced by antithyroid drugs: comparison with idiopathic ANCA vasculitides.抗甲状腺药物诱导的抗中性粒细胞胞浆抗体（ANCA）相关自身免疫性疾病：与特发性ANCA血管炎的比较
Arthritis Res Ther. 2005;7(5):R1072-81. doi: 10.1186/ar1789. Epub 2005 Jul 13.
引用本文的文献
1
Early vasculitis in the mercuric chloride induced Brown Norway rat model is neutrophil independent.在氯化汞诱导的挪威棕色大鼠模型中，早期血管炎与中性粒细胞无关。
Int J Exp Pathol. 1999 Jun;80(3):133-42. doi: 10.1046/j.1365-2613.1999.00113.x.
2
Autoantibodies to myeloperoxidase aggravate mild anti-glomerular-basement-membrane-mediated glomerular injury in the rat.抗髓过氧化物酶自身抗体加重大鼠轻度抗肾小球基底膜介导的肾小球损伤。
Am J Pathol. 1996 Nov;149(5):1695-706.
本文引用的文献
1
Genetic selection for crescent formation yields mouse strain with rapidly progressive glomerulonephritis and small vessel vasculitis.针对新月体形成的基因选择产生了具有快速进展性肾小球肾炎和小血管血管炎的小鼠品系。
Proc Natl Acad Sci U S A. 1993 Apr 15;90(8):3413-7. doi: 10.1073/pnas.90.8.3413.
2
Antimyeloperoxidase-associated proliferative glomerulonephritis: an animal model.抗髓过氧化物酶相关增殖性肾小球肾炎：一种动物模型。
J Exp Med. 1993 Apr 1;177(4):905-14. doi: 10.1084/jem.177.4.905.
3
Further characterization of an animal model of systemic vasculitis.系统性血管炎动物模型的进一步特征研究。
Adv Exp Med Biol. 1993;336:133-7. doi: 10.1007/978-1-4757-9182-2_23.
4
Immune complex glomerulonephritis is induced in rats immunized with heterologous myeloperoxidase.用异种髓过氧化物酶免疫大鼠可诱发免疫复合物性肾小球肾炎。
Clin Exp Immunol. 1994 Sep;97(3):466-73. doi: 10.1111/j.1365-2249.1994.tb06111.x.
5
Anti-neutrophil cytoplasmic antibodies engage and activate human neutrophils via Fc gamma RIIa.抗中性粒细胞胞浆抗体通过FcγRIIa结合并激活人类中性粒细胞。
J Immunol. 1994 Aug 1;153(3):1271-80.
6
Anti-neutrophil cytoplasmic antibodies: current diagnostic and pathophysiological potential.抗中性粒细胞胞浆抗体：当前的诊断及病理生理学潜能
Kidney Int. 1994 Jul;46(1):1-15. doi: 10.1038/ki.1994.239.
7
Cyclosporin A exacerbates mercuric chloride-induced vasculitis in the brown Norway rat.环孢素A会加重氯化汞诱导的棕色挪威大鼠血管炎。
Lab Invest. 1995 Feb;72(2):183-90.
8
Lethal effect of the anti-Fas antibody in mice.抗Fas抗体对小鼠的致死作用。
Nature. 1993 Aug 26;364(6440):806-9. doi: 10.1038/364806a0.
9
10
A spontaneous rheumatoid arthritis-like disease in MRL/l mice.MRL/l小鼠中的一种自发性类风湿性关节炎样疾病。
J Exp Med. 1982 Jun 1;155(6):1690-701. doi: 10.1084/jem.155.6.1690.
Zotero 插件