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The protein C anticoagulant system: inherited defects as basis for venous thrombosis.

作者信息

Dahlbäck B

机构信息

Department of Clinical Chemistry, University of Lund, Malmö General Hospital, Sweden.

出版信息

Thromb Res. 1995 Jan 1;77(1):1-43. doi: 10.1016/0049-3848(94)00138-4.

DOI:10.1016/0049-3848(94)00138-4
PMID:7701473
Abstract
摘要

相似文献

1
The protein C anticoagulant system: inherited defects as basis for venous thrombosis.
Thromb Res. 1995 Jan 1;77(1):1-43. doi: 10.1016/0049-3848(94)00138-4.
2
Factor V Leiden and other coagulation factor mutations affecting thrombotic risk.因子V莱顿突变及其他影响血栓形成风险的凝血因子突变。
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Molecular genetics of familial venous thrombosis.家族性静脉血栓形成的分子遗传学
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Resistance to activated protein C as a basis for venous thrombosis.对活化蛋白C的抵抗作为静脉血栓形成的基础。
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Thrombophilia: the discovery of activated protein C resistance.易栓症：活化蛋白C抵抗的发现。
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The molecular genetics of familial venous thrombosis.家族性静脉血栓形成的分子遗传学
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[Resistance to activated protein C. A common genetic risk factor in venous thrombosis].[对活化蛋白C的抵抗：静脉血栓形成的常见遗传危险因素]
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A novel nonsense mutation in the protein C (PROC) gene (Trp-29-->term) causing recurrent venous thrombosis.蛋白C（PROC）基因中的一种新型无义突变（色氨酸29突变为终止密码子）导致复发性静脉血栓形成。
Hum Genet. 1993 Mar;91(2):196. doi: 10.1007/BF00222726.

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在英国生物银行中，蛋白Z（PROZ）功能丧失与缺血性中风风险增加相关。
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