Stern J H, Meyer D R
Department of Ophthalmology, Albany Medical College, NY 12203, USA.
Ophthalmic Plast Reconstr Surg. 1995 Mar;11(1):49-53. doi: 10.1097/00002341-199503000-00009.
A 24-year-old man sustained orbital and facial injury when an industrial suction device attached to his face. Hemorrhage and edema within the orbital soft tissues were clinically evidenced by proptosis and restricted ocular motility and confirmed by computed tomography. No retinal hemorrhages or exudates were noted. Orbital soft tissue injury caused by sudden extreme decrease in extravascular atmospheric pressure (i.e., orbital barotrauma) is not well described in the ophthalmic literature. The findings in our case are remarkably similar to those seen with traumatic asphyxia and the ophthalmological hydrostatic pressure syndrome, two conditions attributed to an increase in intravascular pressure. Physiologically, our case and these conditions share a similar increase in transmural pressure at the capillary level that is responsible for fluid transudate and hemorrhage in the orbital soft tissues. Intraretinal hemorrhages and exudates typical of Purtscher's retinopathy were conspicuously absent in our patient.
一名24岁男性在工业抽吸装置附着于其面部时，眼眶和面部受伤。眼眶软组织内的出血和水肿在临床上表现为眼球突出和眼球运动受限，并经计算机断层扫描证实。未发现视网膜出血或渗出。眼科学文献中对因血管外大气压力突然急剧下降导致的眼眶软组织损伤（即眼眶气压伤）描述不多。我们病例中的发现与创伤性窒息和眼科静水压力综合征所见极为相似，这两种情况都归因于血管内压力升高。从生理角度来看，我们的病例与这些情况在毛细血管水平上的跨壁压力有类似增加，这是导致眼眶软组织液体渗出和出血的原因。我们的患者明显没有典型的普尔彻视网膜病变的视网膜内出血和渗出。
