Wegmann A, Renker H
Klin Wochenschr. 1976 May 15;54(10):453-9. doi: 10.1007/BF01469045.
Twenty clinical cases described in the literature were gathered, in which electrocardiographic tracings had been recorded. Electrocardiographic changes were found in all cases, such as sinus tachycardia (12 times), impaired electrical conduction (5 times), ventricular overexcitability (7 times), high P-wave (6 times), changes of the ST (10 times), and of the T-wave (18 times). The reason for these changes might be of a complex nature. Based on experimental investiagations mentioned in the discussion, it may be assumed that beside a direct effect on the myocardial cells, the following mechanisms could have influenced the electrical heart potentials: 1. a constriction of the coronary vessels, 2. a pulmonary vascular constriction, 3. an aggregation of thrombocytes, 4. a peripheral vascular dilatation, 5. a bronchiolar constriction.
收集了文献中描述的20例临床病例，这些病例均记录了心电图描记图。所有病例均发现心电图改变，如窦性心动过速（12次）、电传导受损（5次）、心室过度兴奋（7次）、P波高尖（6次）、ST段改变（10次）和T波改变（18次）。这些改变的原因可能很复杂。根据讨论中提及的实验研究，可以假定，除了对心肌细胞的直接作用外，以下机制可能影响心脏电活动：1. 冠状动脉血管收缩；2. 肺血管收缩；3. 血小板聚集；4. 外周血管扩张；5. 细支气管收缩。
