Tumor suppressor genes in molecular medicine.

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作者信息

Hoppe-Seyler F, Butz K

机构信息

Angewandte Tumorvirologie, Deutsches Krebsforschungszentrum, Heidelberg, Germany.

出版信息

Clin Investig. 1994 Aug;72(8):619-30. doi: 10.1007/BF00227456.

DOI:10.1007/BF00227456

PMID:7819720

Abstract

摘要

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The multistep nature of cancer.

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相似文献

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J Mol Med (Berl). 1995 Nov;73(11):529-38. doi: 10.1007/BF00195138.

本文引用的文献

Transcriptional regulation by the retinoblastoma protein.

Trends Cell Biol. 1993 Jul;3(7):211-3. doi: 10.1016/0962-8924(93)90113-f.

In search of the functions of normal p53 protein.

Trends Cell Biol. 1993 Feb;3(2):46-9. doi: 10.1016/0962-8924(93)90151-p.

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Trends Cell Biol. 1993 Feb;3(2):43-6. doi: 10.1016/0962-8924(93)90150-y.

The multistep nature of cancer.

Trends Genet. 1993 Apr;9(4):138-41. doi: 10.1016/0168-9525(93)90209-z.

p53 mutations increase resistance to ionizing radiation.

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A structure-function analysis of transcriptional repression mediated by the WT1, Wilms' tumor suppressor protein.

Oncogene. 1993 Jul;8(7):1713-20.

Integration of cell cycle control with transcriptional regulation by the retinoblastoma protein.

Curr Opin Cell Biol. 1993 Apr;5(2):194-200. doi: 10.1016/0955-0674(93)90102-v.

Molecular characterization of the retinoblastoma susceptibility gene.

Biochim Biophys Acta. 1993 May 25;1155(1):43-61. doi: 10.1016/0304-419x(93)90021-4.

Physical interaction of the retinoblastoma protein with human D cyclins.

Cell. 1993 May 7;73(3):499-511. doi: 10.1016/0092-8674(93)90137-f.

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Annu Rev Med. 1993;44:451-64. doi: 10.1146/annurev.me.44.020193.002315.