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Is amyloid causally involved in pathophysiology of Alzheimer's disease?

作者信息

Saitoh T, Brugge K

机构信息

Department of Neuroscience, School of Medicine, University of California at San Diego, La Jolla 92093.

出版信息

Neurobiol Aging. 1994 Jul-Aug;15(4):461-2. doi: 10.1016/0197-4580(94)90078-7.

DOI:10.1016/0197-4580(94)90078-7
PMID:7969723
Abstract
摘要

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Is amyloid causally involved in pathophysiology of Alzheimer's disease?淀粉样蛋白是否在阿尔茨海默病的病理生理学中起因果作用?
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Amyloid beta-peptide [1-42]-associated free radical-induced oxidative stress and neurodegeneration in Alzheimer's disease brain: mechanisms and consequences.淀粉样β肽[1-42]相关自由基诱导的氧化应激与阿尔茨海默病脑内神经退行性变:机制与后果
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The possible origin of free radicals from amyloid beta peptides in Alzheimer's disease.
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Free radicals, proteolysis, and the degeneration of neurons in Alzheimer disease: how essential is the beta-amyloid link?自由基、蛋白水解作用与阿尔茨海默病中神经元的退化:β-淀粉样蛋白的联系有多重要?
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beta-Amyloid formation as a potential therapeutic target for Alzheimer's disease.β-淀粉样蛋白的形成作为阿尔茨海默病的潜在治疗靶点。
Annu Rev Pharmacol Toxicol. 1994;34:69-89. doi: 10.1146/annurev.pa.34.040194.000441.
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The Sixth International Conference on Alzheimer's disease, Amsterdam, The Netherlands, July 1998. The amyloid cascade hypothesis debate: emerging consensus on the role of A beta and amyloid in Alzheimer's disease.第六届阿尔茨海默病国际会议,荷兰阿姆斯特丹,1998年7月。淀粉样蛋白级联假说辩论:关于β淀粉样蛋白(Aβ)和淀粉样蛋白在阿尔茨海默病中作用的新共识。
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J Neural Transm (Vienna). 2000;107(2):203-31. doi: 10.1007/s007020050018.

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