热休克诱导潜伏感染的小鼠三叉神经节细胞在解离培养物中单纯疱疹病毒1型的重新激活。
Heat shock-induced reactivation of herpes simplex virus type 1 in latently infected mouse trigeminal ganglion cells in dissociated culture.
作者信息
Moriya A, Yoshiki A, Kita M, Fushiki S, Imanishi J
机构信息
Department of Microbiology, Kyoto Prefectural University of Medicine, Japan.
出版信息
Arch Virol. 1994;135(3-4):419-25. doi: 10.1007/BF01310025.
PMID:7979977
Abstract
An in vitro herpes simplex virus type 1 (HSV-1) latency model was established, using trigeminal ganglia from latently infected mice. When heat-treated at 43 degrees C for 3 h, reactivation followed in 76.6% of the cultures, while reactivation was not observed in non-heat-treated controls.
摘要
利用来自潜伏感染小鼠的三叉神经节建立了一种1型单纯疱疹病毒（HSV-1）体外潜伏模型。当在43℃热处理3小时后，76.6%的培养物出现了再激活，而在未热处理的对照中未观察到再激活。
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