Mapping of an insulin-dependent diabetes locus, Idd9, in NOD mice to chromosome 4.

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作者信息

Rodrigues N R, Cornall R J, Chandler P, Simpson E, Wicker L S, Peterson L B, Todd J A

机构信息

Nuffield Department of Surgery, University of Oxford, John Radcliffe Hospital, Headington, UK.

出版信息

Mamm Genome. 1994 Mar;5(3):167-70. doi: 10.1007/BF00352349.

DOI:10.1007/BF00352349

PMID:8199406

Abstract

摘要

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本文引用的文献

Polygenic control of autoimmune diabetes in nonobese diabetic mice.非肥胖糖尿病小鼠自身免疫性糖尿病的多基因控制

Nat Genet. 1993 Aug;4(4):404-9. doi: 10.1038/ng0893-404.

Statistical evaluation of multiple-locus linkage data in experimental species and its relevance to human studies: application to nonobese diabetic (NOD) mouse and human insulin-dependent diabetes mellitus (IDDM).实验物种多位点连锁数据的统计学评估及其与人类研究的相关性：应用于非肥胖糖尿病（NOD）小鼠和人类胰岛素依赖型糖尿病（IDDM）

Am J Hum Genet. 1993 Sep;53(3):702-14.

Promotion of spontaneous diabetes in non-obese diabetes-prone mice by cyclophosphamide.环磷酰胺促进非肥胖糖尿病易患小鼠自发性糖尿病的发生

Diabetologia. 1984 Dec;27(6):604-6. doi: 10.1007/BF00276978.

Genetic control of diabetes and insulitis in the nonobese diabetic (NOD) mouse.非肥胖糖尿病（NOD）小鼠中糖尿病和胰岛炎的遗传控制。

J Exp Med. 1987 Jun 1;165(6):1639-54. doi: 10.1084/jem.165.6.1639.

Assessing the role of HLA-linked and unlinked determinants of disease.评估HLA相关和非相关疾病决定因素的作用。

Am J Hum Genet. 1987 Jan;40(1):1-14.

The NOD mouse: recessive diabetogenic gene in the major histocompatibility complex.非肥胖型糖尿病（NOD）小鼠：主要组织相容性复合体中的隐性致糖尿病基因。

Science. 1986 Feb 14;231(4739):733-5. doi: 10.1126/science.3003909.

Anti-suppressor effect of cyclophosphamide on the development of spontaneous diabetes in NOD mice.环磷酰胺对非肥胖糖尿病（NOD）小鼠自发性糖尿病发展的抗抑制作用。

Eur J Immunol. 1988 Mar;18(3):481-4. doi: 10.1002/eji.1830180325.

Three recessive loci required for insulin-dependent diabetes in nonobese diabetic mice.非肥胖糖尿病小鼠中胰岛素依赖型糖尿病所需的三个隐性基因座。

Science. 1987 Jul 17;237(4812):286-9. doi: 10.1126/science.2885918.

A note on multiple testing procedures in linkage analysis.关于连锁分析中多重检验程序的一则注释。

Am J Hum Genet. 1991 Jun;48(6):1058-64.

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Nature. 1991 Nov 14;354(6349):155-9. doi: 10.1038/354155a0.

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