Lapp N L, Morgan W K, Zaldivar G
Pulmonary Division, West Virginia University Medical Center, University Hospital, Morgantown 26506.
Occup Environ Med. 1994 Apr;51(4):234-8. doi: 10.1136/oem.51.4.234.
It has recently been suggested that the inhalation of coal in the absence of complicated coal workers' pneumoconiosis (CWP) or smoking can lead to disabling airways obstruction. The cause of such obstruction has been variously attributed to emphysema or bronchitis. The frequency of significant airways obstruction in a group of United States coal miners seeking compensation for occupationally induced pulmonary impairment was therefore determined. In a sample of 611 "Black Lung" claimants there was only one subject who was a non-smoker and who in the absence of other non-occupationally related diseases,--for example, asthma and bronchiectasis--had sufficient airways obstruction to render it difficult for him to carry out hard labour. An alternative explanation for his reduced ventilatory capacity other than coal dust or smoking may be available. If the inhalation of coal dust in the absence of smoking and complicated CWP ever induces sufficient ventilatory impairment to preclude a miner from working, it is indeed rare.
最近有人提出,在没有复杂的煤工尘肺(CWP)或吸烟的情况下吸入煤尘会导致致残性气道阻塞。这种阻塞的原因被多种归因于肺气肿或支气管炎。因此,确定了一组因职业性肺损伤寻求赔偿的美国煤矿工人中严重气道阻塞的发生率。在611名“黑肺病”索赔者的样本中,只有一名非吸烟者,在没有其他与非职业相关疾病(如哮喘和支气管扩张)的情况下,有足够的气道阻塞,使他难以从事繁重劳动。除了煤尘或吸烟外,可能还有其他原因导致他的通气能力下降。如果在不吸烟和没有复杂CWP的情况下吸入煤尘确实会导致足够的通气功能损害,使矿工无法工作,那确实是罕见的。