Ovsianikov V I, Aleksandrov A N
Fiziol Zh Im I M Sechenova. 1993 Jun;79(6):133-41.
Jb been shown that beta-adrenergic stimulation by isadrine can lead to decreasing as well increasing of spre activity of the pyloric sphincter and small intestine administration of isadrine in conditions of the blockade of M- and N-cholinoreceptors suppressed the spike activity in all the portions of the intestine. The data obtained suggest that excitatory beta-adrenoreceptors are located on the cholinergic interneurons of the enteral nervous system. The suppression of the spike activity was, evidently, due to a stimulation of postsynaptic beta-adrenoreceptors.
已表明，异丙肾上腺素对β-肾上腺素能的刺激可导致幽门括约肌和小肠运动活性的降低以及增加。在M和N胆碱能受体被阻断的情况下给予异丙肾上腺素，可抑制肠道所有部位的锋电位活动。所获得的数据表明，兴奋性β-肾上腺素能受体位于肠神经系统的胆碱能中间神经元上。锋电位活动的抑制显然是由于对突触后β-肾上腺素能受体的刺激所致。
