Hausmanowa-Petrusewicz I, Emeryk B, Rowińska-Marcińska K, Jedrzejowska H
J Neurol. 1979 May 2;220(3):169-84. doi: 10.1007/BF00705535.
Fifty cases of the Guillain-Barré-Strohl syndrome were investigated clinically and electrophysiologically--20 in the acute phase, and 30, as a matter of followup, many years after. The sural nerve was biopsied in six cases. There was no evident correlation between clinical symptoms and slowing of motor and sensory conduction. Nerve conduction velocity became slower after the beginning of clinical improvement. The electrophysiological abnormalities concerned both sensory and motor fibers despite the frequent absence of clinical sensory manifestations. The so-called long nerves were involved earlier and more markedly than the so-called short nerves. Conduction velocity and distal latency were equally affected. A slight electrophysiological defect was noticeable even many years after the acute phase of the syndrome, in completely symptoms free patients. Some correlation existed between conduction velocity changes and histological findings.
对50例吉兰-巴雷-施特罗尔综合征患者进行了临床和电生理研究——急性期20例，作为随访，多年后30例。对6例患者进行了腓肠神经活检。临床症状与运动和感觉传导减慢之间无明显相关性。临床症状开始改善后神经传导速度变慢。尽管临床上经常没有感觉表现，但电生理异常涉及感觉和运动纤维。所谓的长神经比所谓的短神经更早、更明显地受累。传导速度和远端潜伏期同样受到影响。即使在综合征急性期多年后，在完全无症状的患者中也可注意到轻微的电生理缺陷。传导速度变化与组织学结果之间存在一定相关性。
