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精神分裂症能否用多汉关于基因与饮食中肽超载相互作用的假说来合理解释?

Could schizophrenia be reasonably explained by Dohan's hypothesis on genetic interaction with a dietary peptide overload?

作者信息

Reichelt K L, Seim A R, Reichelt W H

机构信息

Institute of Pediatric Research, Univ of Oslo, Rikshopsitalet, Norway.

出版信息

Prog Neuropsychopharmacol Biol Psychiatry. 1996 Oct;20(7):1083-114. doi: 10.1016/s0278-5846(96)00099-1.

Abstract
  1. Dohan has proposed that schizophrenia is a genetic disposition which interacts with an overload of dietary proteins such as casein and gluten or gliadin. 2. A systematic attempt is made to see if this hypothesis is possible faced with aspects of schizophrenia that must be accounted for. 3. The authors conclusion is that it is possible, but more serious work in this field is urgently needed.
摘要
  1. 多汉提出,精神分裂症是一种遗传倾向,它与酪蛋白、麸质或麦醇溶蛋白等膳食蛋白质过量相互作用。2. 我们进行了系统的尝试,以探讨面对精神分裂症必须考虑的各个方面时,这一假设是否成立。3. 作者的结论是,这是有可能的,但该领域迫切需要开展更深入的研究。

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