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生物反馈诱导严重支气管哮喘患者自主降低呼吸阻力

Biofeedback-induced voluntary reduction of respiratory resistance in severe bronchial asthma.

作者信息

Mass R, Richter R, Dahme B

机构信息

University Hospital Hamburg-Eppendorf, Psychiatric Clinic, Germany.

出版信息

Behav Res Ther. 1996 Oct;34(10):815-9. doi: 10.1016/0005-7967(96)00063-0.

Abstract

Attempts to reduce asthmatic symptoms with respiratory resistance (Ros) biofeedback yielded heterogeneous results. The nature of treatment effects remained unclear (e.g. unspecific relaxation, visceral learning); little is known about clinical long-term effects. The present study investigated the suitability of a statistical single-case approach for an adequate evaluation. A female asthmatic underwent 12 Ros biofeedback sessions. Several breathing parameters were recorded during all sessions and pre/post investigations. The clinical course was observed with symptom diaries for 100 days. The subject achieved considerable voluntary control over her Ros, this control was based upon the expiratory flow. In general, the effects on the clinical course were weak; however, asthmatic attacks decreased in the follow-up period. Careful analyses of physiological, psychophysiological, and course variables allowed the identification of the main processes underlying the biofeedback effects. Further studies should investigate the worth of the expiratory flow as feedback variable.

摘要

尝试通过呼吸阻力(Ros)生物反馈来减轻哮喘症状,结果参差不齐。治疗效果的本质仍不明确(例如非特异性放松、内脏学习);关于临床长期效果知之甚少。本研究调查了一种统计单病例方法用于充分评估的适用性。一名女性哮喘患者接受了12次Ros生物反馈治疗。在所有治疗期间以及治疗前/后调查中记录了几个呼吸参数。通过症状日记观察临床病程100天。该受试者对其Ros实现了相当程度的自主控制,这种控制基于呼气流量。总体而言,对临床病程的影响较弱;然而,在随访期间哮喘发作次数减少。对生理、心理生理和病程变量的仔细分析有助于确定生物反馈效果背后的主要过程。进一步的研究应调查呼气流量作为反馈变量的价值。

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