Helveston E M, Mora J S, Lipsky S N, Plager D A, Ellis F D, Sprunger D T, Sondhi N
Department of Ophthalmology, Indiana University Medical School, Indianapolis, USA.
Trans Am Ophthalmol Soc. 1996;94:315-28; discussion 328-34.
Reports of several large series of patients with superior oblique palsy (SOP) published in 1986 or before set forth important guidelines for both diagnosis and treatment of this condition. Newer information about the anatomy, physiology, and pathophysiology of the superior oblique has accrued over the past decade. This paper reviews our experience with diagnosis and treatment of SOP over the past 5 years in light of this new information. Charts of patients treated for SOP over 5 year (1990 to 1995) were reviewed for male or female sex, age, symptoms, refraction, vision, stereo acuity, head posture, facial asymmetry, intraoperative superior oblique traction test, diagnostic position prism and cover test, torsion, surgery performed, and results of treatment. The charts of 190 patients were reviewed. In 181, postoperative examinations were performed by us. The etiology of the SOP was congenital in 137 and acquired in 53. Twenty-nine acquired cases were due to trauma and 24 arose from other causes. Fifty-six patients had facial asymmetry, 51 of whom had congenital SOP. Ninety-five had a lax tendon, 83 (87%) of whom had congenital SOP. Sixty-six had a normal tendon, 29 (44%) of whom had acquired SOP. Seventy-seven percent of patients had Knapp class I, III, or IV palsy. An average of 1.26 surgeries was performed per patient. Inferior oblique weakening was performed in 177 (93%), while 68 vertical rectus recessions were done. Thirty-five patients had superior oblique tuck or resection, all on lax tendons, and 15 had Harada Ito procedures for torsion. Six patients had mild Brown syndrome postoperatively, none of which required a takedown. A cure, defined as relief of symptoms or elimination of strabismus and head tilt, was achieved in 166 of 181 (92%) of patients. Successful treatment of SOP can be accomplished in the majority of cases by selective surgery usually beginning with inferior oblique weakening plus additional vertical rectus and horizontal surgery as needed, with superior oblique strengthening used only for lax tendons or when torsion is the main problem.
1986年或更早发表的几篇关于上斜肌麻痹(SOP)的大型患者系列报告为该病症的诊断和治疗提出了重要指导方针。在过去十年中,积累了有关上斜肌解剖学、生理学和病理生理学的新信息。本文根据这些新信息回顾了我们在过去5年中对SOP的诊断和治疗经验。回顾了1990年至1995年5年间接受SOP治疗患者的病历,包括性别、年龄、症状、屈光、视力、立体视锐度、头位、面部不对称、术中上斜肌牵引试验、诊断性三棱镜和遮盖试验、扭转、所施行的手术以及治疗结果。共回顾了190例患者的病历。其中181例患者接受了术后检查。SOP的病因先天性的有137例,后天性的有53例。29例后天性病例由外伤引起,24例由其他原因引起。56例患者存在面部不对称,其中51例为先天性SOP。95例患者肌腱松弛,其中83例(87%)为先天性SOP。66例患者肌腱正常,其中29例(44%)为后天性SOP。77%的患者为Knapp I、III或IV级麻痹。每位患者平均接受1.26次手术。177例(93%)患者进行了下斜肌减弱术,同时进行了68例垂直直肌后徙术。35例患者进行了上斜肌折叠或切除术,均针对松弛肌腱,15例患者进行了用于扭转的原田伊藤手术。6例患者术后出现轻度布朗综合征,均无需拆除手术。181例患者中有166例(92%)症状缓解或斜视和头位偏斜消除,达到治愈标准。大多数情况下,通过选择性手术通常先进行下斜肌减弱术,并根据需要附加垂直直肌和水平手术,仅在肌腱松弛或扭转是主要问题时使用上斜肌加强术,即可成功治疗SOP。