非HLA基因与胰岛素依赖型糖尿病易感性：CTLA-4基因的作用 - Suppr

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超能文献

Non-HLA genes and the susceptibility to insulin dependent diabetes: the role of the CTLA-4 gene.

作者信息

Pozzilli P, Buzzetti R, Nisticó L, Romiti A, Giovannini C

机构信息

Istituto di Clinica Medica II, Universitá di Roma, La Sapienza, Italy.

出版信息

Acta Diabetol. 1996 Dec;33(4):250-2. doi: 10.1007/BF00571558.

DOI:10.1007/BF00571558

PMID:9033962

Abstract

摘要

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Non-HLA genes and the susceptibility to insulin dependent diabetes: the role of the CTLA-4 gene.非HLA基因与胰岛素依赖型糖尿病易感性：CTLA-4基因的作用

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CTLA4 gene haplotypes cannot protect from IDDM in the presence of high-risk HLA DQ8 or DQ2 alleles in German families.在德国家庭中，若存在高危HLA DQ8或DQ2等位基因，CTLA4基因单倍型无法预防胰岛素依赖型糖尿病。

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本文引用的文献

The CTLA-4 gene region of chromosome 2q33 is linked to, and associated with, type 1 diabetes. Belgian Diabetes Registry.位于2号染色体q33区域的细胞毒性T淋巴细胞相关抗原4（CTLA-4）基因与1型糖尿病相关联。比利时糖尿病登记处。

Hum Mol Genet. 1996 Jul;5(7):1075-80. doi: 10.1093/hmg/5.7.1075.

Confirmation of three susceptibility genes to insulin-dependent diabetes mellitus: IDDM4, IDDM5 and IDDM8.胰岛素依赖型糖尿病三个易感基因的确认：IDDM4、IDDM5和IDDM8。

Hum Mol Genet. 1996 May;5(5):693-8. doi: 10.1093/hmg/5.5.693.

The Th1/Th2 balance in autoimmunity.自身免疫中的Th1/Th2平衡。

Curr Opin Immunol. 1995 Dec;7(6):793-8. doi: 10.1016/0952-7915(95)80050-6.

Genetic analysis of immune dysfunction in non-obese diabetic (NOD) mice: mapping of a susceptibility locus close to the Bcl-2 gene correlates with increased resistance of NOD T cells to apoptosis induction.非肥胖型糖尿病（NOD）小鼠免疫功能障碍的遗传分析：与Bcl-2基因邻近的一个易感位点的定位与NOD T细胞对凋亡诱导的抗性增加相关。

Eur J Immunol. 1994 Feb;24(2):380-4. doi: 10.1002/eji.1830240217.

A genome-wide search for human type 1 diabetes susceptibility genes.一项针对人类1型糖尿病易感基因的全基因组搜索。

Nature. 1994 Sep 8;371(6493):130-6. doi: 10.1038/371130a0.

Multiple DNA variant association analysis: application to the insulin gene region in type I diabetes.多重DNA变异关联分析：在I型糖尿病胰岛素基因区域的应用

Am J Hum Genet. 1994 Dec;55(6):1247-54.

Lawrence Lecture. The Emperor's new genes: 1993 RD Lawrence Lecture.劳伦斯讲座。皇帝的新基因：1993年RD劳伦斯讲座。

Diabet Med. 1994 Jan-Feb;11(1):6-16. doi: 10.1111/j.1464-5491.1994.tb00222.x.

CTLA-4 can function as a negative regulator of T cell activation.细胞毒性T淋巴细胞相关抗原4（CTLA-4）可作为T细胞活化的负调节因子发挥作用。

Immunity. 1994 Aug;1(5):405-13. doi: 10.1016/1074-7613(94)90071-x.

Positional cloning works! Identification of genes that cause IDDM.

Diabetes. 1995 Feb;44(2):139-40. doi: 10.2337/diab.44.2.139.

CTLA-4 gene polymorphism associated with Graves' disease in a Caucasian population.CTLA-4基因多态性与高加索人群中的格雷夫斯病相关。

J Clin Endocrinol Metab. 1995 Jan;80(1):41-5. doi: 10.1210/jcem.80.1.7829637.

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