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Role of macrophages in immunity and pathogenesis of experimental cryptococcosis induced by the airborne route.--Part I: Pathogenesis and acquired immunity of Cryptococcus neoformans.

作者信息

Karaoui R M, Hall N K, Larsh H W

出版信息

Mykosen. 1977 Oct;20(10):380-8.

PMID:927451
Abstract
摘要

相似文献

1
Role of macrophages in immunity and pathogenesis of experimental cryptococcosis induced by the airborne route.--Part I: Pathogenesis and acquired immunity of Cryptococcus neoformans.巨噬细胞在经空气传播途径诱导的实验性隐球菌病免疫及发病机制中的作用。——第一部分:新型隐球菌的发病机制与获得性免疫。
Mykosen. 1977 Oct;20(10):380-8.
2
Role of macrophages in immunity and pathogenesis of experimental cryptococcosis induced by the airborne route--Part II: Phagocytosis and intracellular fate of Cryptococcus neoformans.巨噬细胞在经空气传播途径诱导的实验性隐球菌病免疫及发病机制中的作用——第二部分:新型隐球菌的吞噬作用及细胞内命运
Mykosen. 1977 Nov;20(11):409-12.
3
Evaluation of host immune responses to pulmonary cryptococcosis using a temperature-sensitive C. neoformans calcineurin A mutant strain.使用温度敏感型新型隐球菌钙调神经磷酸酶A突变株评估宿主对肺隐球菌病的免疫反应。
Microb Pathog. 2005 Feb-Mar;38(2-3):113-23. doi: 10.1016/j.micpath.2004.12.007.
4
Afferent phase production of TNF-alpha is required for the development of protective T cell immunity to Cryptococcus neoformans.产生肿瘤坏死因子-α的传入相对于新型隐球菌保护性T细胞免疫的发展是必需的。
J Immunol. 1996 Nov 15;157(10):4529-36.
5
Immunization of mice with a mutant of Cryptococcus neoformans. Characterization of the mutant, actively acquired resistance to experimental cryptococcosis in mice.用新型隐球菌突变体对小鼠进行免疫接种。突变体的特性,小鼠对实验性隐球菌病的主动获得性抗性。
Dermatologica. 1976;152(1):16-22.
6
Cutting edge: Role of C-C chemokine receptor 5 in organ-specific and innate immunity to Cryptococcus neoformans.前沿:C-C趋化因子受体5在针对新型隐球菌的器官特异性免疫和固有免疫中的作用
J Immunol. 1999 Nov 1;163(9):4642-6.
7
CD4+ T cell-dependent acquired state of immunity that protects the brain against Cryptococcus neoformans.依赖CD4 + T细胞的获得性免疫状态,可保护大脑免受新型隐球菌的侵害。
J Immunol. 1994 Mar 1;152(5):2344-50.
8
The role of monocyte chemotactic protein-1 (MCP-1) in the recruitment of monocytes and CD4+ T cells during a pulmonary Cryptococcus neoformans infection.单核细胞趋化蛋白-1(MCP-1)在肺部新型隐球菌感染期间单核细胞和CD4 + T细胞募集中的作用。
J Immunol. 1995 Nov 15;155(10):4790-7.
9
Immunization of mice with stable, acapsular, yeast-like mutants of Cryptococcus neoformans.用新型隐球菌稳定的、无荚膜的酵母样突变体对小鼠进行免疫接种。
Sabouraudia. 1983 Jun;21(2):113-9.
10
Neutropenia alters lung cytokine production in mice and reduces their susceptibility to pulmonary cryptococcosis.中性粒细胞减少会改变小鼠肺部细胞因子的产生,并降低它们对肺部隐球菌病的易感性。
Eur J Immunol. 2003 Jun;33(6):1744-53. doi: 10.1002/eji.200323626.

引用本文的文献

1
The regulation of pulmonary immunity.肺部免疫的调节
Adv Immunol. 1995;59:369-455. doi: 10.1016/s0065-2776(08)60634-3.
2
Immunity in cryptococcosis: an overview.隐球菌病中的免疫:概述
Mycopathologia. 1982 Mar 19;77(3):183-90. doi: 10.1007/BF00518804.
3
Host-etiological agent interactions in intranasally and intraperitoneally induced Cryptococcosis in mice.小鼠经鼻内和腹腔内诱导隐球菌病中宿主与病原体的相互作用
Infect Immun. 1980 Aug;29(2):633-41. doi: 10.1128/iai.29.2.633-641.1980.
4
Experimental cryptococcosis in guinea pigs.豚鼠实验性隐球菌病
Mycopathologia. 1983 Jun 20;82(3):179-84. doi: 10.1007/BF00439224.
5
An overview of macrophage-fungal interactions.巨噬细胞与真菌相互作用概述。
Mycopathologia. 1986 Feb;93(2):77-93. doi: 10.1007/BF00437738.
6
Persistence of infection in mice inoculated intranasally with Cryptococcus neoformans.经鼻内接种新型隐球菌的小鼠体内感染的持续性。
Mycopathologia. 1988 Dec;104(3):163-9. doi: 10.1007/BF00437432.
7
CD4+ T cells cause multinucleated giant cells to form around Cryptococcus neoformans and confine the yeast within the primary site of infection in the respiratory tract.CD4+ T细胞促使多核巨细胞在新型隐球菌周围形成,并将酵母限制在呼吸道感染的原发部位。
J Exp Med. 1992 Jun 1;175(6):1685-95. doi: 10.1084/jem.175.6.1685.
8
Immunization of mice with an avirulent pseudohyphal form of Cryptococcus neoformans.用新型隐球菌无毒假菌丝形式对小鼠进行免疫接种。
Mycopathologia. 1979 Sep 28;68(3):179-81. doi: 10.1007/BF00578527.
9
Kinetics of lymphocyte transformation in mice immunized with viable avirulent forms of Cryptococcus neoformans.用新型隐球菌活无毒株免疫的小鼠淋巴细胞转化动力学
Infect Immun. 1979 May;24(2):449-53. doi: 10.1128/iai.24.2.449-453.1979.