Berny P J, Buronfosse T, Buronfosse F, Lamarque F, Lorgue G
Dépt. SBFA, Laboratoire de Toxicologie, ENVL, Marcy l'Etoile, France.
Chemosphere. 1997 Oct;35(8):1817-29. doi: 10.1016/s0045-6535(97)00242-7.
This paper presents the result of a 4 year survey in France (1991-1994) based on the activity of a wildlife disease surveillance network (SAGIR). The purpose of this study was to evaluate the detrimental effects of anticoagulant (Ac) rodenticides in non-target wild animals. Ac poisoning accounted for a very limited number of the identified causes of death (1-3%) in most species. Predators (mainly foxes and buzzards) were potentially exposed to anticoagulant compounds (especially bromadiolone) via contaminated prey in some instances. The liver concentrations of bromadiolone residues were elevated and species-specific diagnostic values were determined. These values were quite similar to those reported in the literature when secondary anticoagulant poisoning was experimentally assessed.
本文介绍了基于野生动物疾病监测网络（SAGIR）的活动在法国进行的为期4年（1991 - 1994年）的调查结果。本研究的目的是评估抗凝血灭鼠剂对非目标野生动物的有害影响。在大多数物种中，抗凝血剂中毒在已确定的死亡原因中占比非常有限（1% - 3%）。在某些情况下，食肉动物（主要是狐狸和秃鹰）可能通过受污染的猎物接触到抗凝血化合物（尤其是溴敌隆）。测定了肝脏中溴敌隆残留浓度以及物种特异性诊断值。这些值与文献中在实验评估继发性抗凝血剂中毒时报告的值非常相似。
