Sundén-Cullberg J, Tedroff J, Aquilonius S M
Department of Neurology, University Hospital, Uppsala, Sweden.
Mov Disord. 1998 Jan;13(1):147-9. doi: 10.1002/mds.870130127.
A 20-year-old woman with acute chorea induced by primary antiphospholipid syndrome was studied by using fluorodeoxyglucose and positron emission tomography (PET). PET sessions were conducted during an episode of severe chorea and after recovery. The symptoms predominantly affected the right side of her face and body, and PET demonstrated a corresponding increase in lentiform and caudate nucleus metabolism prevailing on the left side. After recovery, PET showed normal values in the regions previously studied. This study adds further evidence to support the theory that acute choreas are somehow the result of striatal hypermetabolism.
一名20岁因原发性抗磷脂综合征诱发急性舞蹈症的女性，通过使用氟脱氧葡萄糖和正电子发射断层扫描（PET）进行了研究。PET检查在严重舞蹈症发作期间及恢复后进行。症状主要影响她面部和身体的右侧，PET显示左侧豆状核和尾状核代谢相应增加。恢复后，PET显示先前研究区域的值正常。这项研究进一步证明了急性舞蹈症在某种程度上是纹状体代谢亢进的结果这一理论。
