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米勒肌作用的重新审视。

The role of Müller's muscle reconsidered.

作者信息

Bang Y H, Park S H, Kim J H, Cho J H, Lee C J, Roh T S

机构信息

Department of Plastic Surgery at Inha General Hospital, Songnam, Korea.

出版信息

Plast Reconstr Surg. 1998 Apr;101(5):1200-4. doi: 10.1097/00006534-199804050-00004.

Abstract

It is a traditional teaching that the levator aponeurosis is the main transmitter of the levator palpebrae muscle. However, there are several points that raise doubts in this fundamental concept of the levator aponeurosis as being the primary interconnecting mechanism in upper lid elevation. Despite the structural integrity of the levator complex, drooping of the upper eyelids is seen to develop in situations such as Horner's syndrome and in times of excessive fatigue and sleepiness. Amid the controversy in the literature regarding the specific role of the levator aponeurosis in the lid-elevating mechanism, we have observed that the levator aponeurosis fails to make constant attachment to the tarsal plate. This has led us to speculate on the possible role of the posterior lamella - Müller's muscle - as the primary transmitter of the levator muscle action to the tarsal plate.

摘要

传统观点认为提上睑肌腱膜是提上睑肌的主要传导结构。然而,在这一提上睑肌腱膜作为上睑提升主要连接机制的基本概念中,有几点令人质疑。尽管提上睑肌复合体结构完整,但在霍纳综合征以及过度疲劳和困倦等情况下,仍会出现上睑下垂。在关于提上睑肌腱膜在眼睑提升机制中具体作用的文献争议中,我们观察到提上睑肌腱膜未能持续附着于睑板。这使我们推测后层(米勒肌)可能作为提上睑肌作用于睑板的主要传导结构。

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