脓毒症中的心脏功能障碍：新理论与临床意义 - Suppr

Suppr

超能文献

Cardiac dysfunction in sepsis: new theories and clinical implications.

作者信息

Grocott-Mason R M, Shah A M

机构信息

Department of Cardiology, University of Wales College of Medicine, Cardiff, UK.

出版信息

Intensive Care Med. 1998 Apr;24(4):286-95. doi: 10.1007/s001340050570.

DOI:10.1007/s001340050570

PMID:9609405

Abstract

摘要

相似文献

Cardiac dysfunction in sepsis: new theories and clinical implications.

Intensive Care Med. 1998 Apr;24(4):286-95. doi: 10.1007/s001340050570.

Cardiac dysfunction in septic shock and multiple organ dysfunction syndrome.

Crit Care Nurs Q. 1994 Feb;16(4):39-48. doi: 10.1097/00002727-199402000-00006.

Cardiac dysfunction in severe sepsis and septic shock.

Curr Opin Crit Care. 2009 Oct;15(5):392-7. doi: 10.1097/MCC.0b013e3283307a4e.

Myocardial dysfunction in sepsis. Recent insights.

Chest. 1989 May;95(5):941-5. doi: 10.1378/chest.95.5.941.

Hemophagocytic lymphohistiocytosis mimicking septic shock.

Chest. 2012 Mar;141(3):835. doi: 10.1378/chest.11-2717.

[Acute septic cardiomyopathy].

Internist (Berl). 1998 May;39(5):467-78. doi: 10.1007/s001080050198.

[Diastolic malfunction in sepsis and septic shock].

Anesteziol Reanimatol. 2004 Jul-Aug(4):47-9.

Is cell death a prerequisite for cardiac dysfunction during sepsis?

Crit Care Med. 2005 May;33(5):1160-2. doi: 10.1097/01.ccm.0000159727.27059.d2.

Critical illness polyneuropathy in patients with systemic inflammatory response syndrome or septic shock.

Intensive Care Med. 2001 Mar;27(3):613. doi: 10.1007/s001340100850.

Cardiovascular dysfunction in sepsis and critical illness.

Vet Clin North Am Small Anim Pract. 2011 Jul;41(4):717-26, v. doi: 10.1016/j.cvsm.2011.04.003. Epub 2011 Jun 16.

引用本文的文献

Integrated analysis of multi-omics data reveals T cell exhaustion in sepsis.

Front Immunol. 2023 Apr 3;14:1110070. doi: 10.3389/fimmu.2023.1110070. eCollection 2023.

Prognostic tools for elderly patients with sepsis: in search of new predictive models.

Intern Emerg Med. 2021 Jun;16(4):1027-1030. doi: 10.1007/s11739-021-02729-5. Epub 2021 Apr 13.

Stress cardiomyopathy of the critically ill: Spectrum of secondary, global, probable and subclinical forms.

Indian Heart J. 2018 Jan-Feb;70(1):177-184. doi: 10.1016/j.ihj.2017.04.005. Epub 2017 Apr 19.

Role of TFEB Mediated Autophagy, Oxidative Stress, Inflammation, and Cell Death in Endotoxin Induced Myocardial Toxicity of Young and Aged Mice.

Oxid Med Cell Longev. 2016;2016:5380319. doi: 10.1155/2016/5380319. Epub 2016 Apr 21.

Hyperdynamic left ventricular ejection fraction in the intensive care unit.

Crit Care. 2015 Aug 7;19(1):288. doi: 10.1186/s13054-015-1012-8.

Propofol infusion syndrome.

Forensic Sci Med Pathol. 2006 Dec;2(4):277-81. doi: 10.1385/FSMP:2:4:277.

Prognostic value of troponins in sepsis: a meta-analysis.

Intensive Care Med. 2013 Jul;39(7):1181-9. doi: 10.1007/s00134-013-2902-3. Epub 2013 Apr 18.

Phospholemman: a novel cardiac stress protein.

Clin Transl Sci. 2010 Aug;3(4):189-96. doi: 10.1111/j.1752-8062.2010.00213.x.

Sepsis-induced cardiomyopathy: a review of pathophysiologic mechanisms.

Heart Fail Rev. 2010 Nov;15(6):605-11. doi: 10.1007/s10741-010-9176-4.

Protein phosphatase 2A contributes to the cardiac dysfunction induced by endotoxemia.

Cardiovasc Res. 2009 Apr 1;82(1):67-76. doi: 10.1093/cvr/cvp037. Epub 2009 Feb 6.

本文引用的文献

Basal release of nitric oxide augments the Frank-Starling response in the isolated heart.

Circulation. 1997 Aug 19;96(4):1320-9. doi: 10.1161/01.cir.96.4.1320.

Nitric oxide synthase inhibition by L-NAME in leukocytopenic patients with severe septic shock.

Intensive Care Med. 1997 May;23(5):561-6. doi: 10.1007/s001340050373.

Left ventricular systolic and diastolic function in septic shock.

Intensive Care Med. 1997 May;23(5):553-60. doi: 10.1007/s001340050372.

Nitric oxide can increase heart rate by stimulating the hyperpolarization-activated inward current, I(f).

Circ Res. 1997 Jul;81(1):60-8. doi: 10.1161/01.res.81.1.60.

Leukocytes and decreased left-ventricular contractility during endotoxemia in rabbits.

Am J Respir Crit Care Med. 1997 Jun;155(6):1977-83. doi: 10.1164/ajrccm.155.6.9196105.

Cytokines, nitrite/nitrate, soluble tumor necrosis factor receptors, and procalcitonin concentrations: comparisons in patients with septic shock, cardiogenic shock, and bacterial pneumonia.

Crit Care Med. 1997 Apr;25(4):607-13. doi: 10.1097/00003246-199704000-00009.

Partial inhibition of protein synthesis by Pseudomonas exotoxin A deranges catecholamine sensitivity of cultured rat heart myocytes.

J Mol Cell Cardiol. 1997 Feb;29(2):799-811. doi: 10.1006/jmcc.1996.0324.

Inhibition of myocardial crossbridge cycling by hypoxic endothelial cells: a potential mechanism for matching oxygen supply and demand?

Circ Res. 1997 May;80(5):688-98. doi: 10.1161/01.res.80.5.688.

L-canavanine, an inhibitor of inducible nitric oxide synthase, improves venous return in endotoxemic rats.

Crit Care Med. 1997 Mar;25(3):469-75. doi: 10.1097/00003246-199703000-00016.

Inducible nitric oxide synthase and tumor necrosis factor-alpha in myocardium in human dilated cardiomyopathy.

J Am Coll Cardiol. 1997 Mar 15;29(4):716-24. doi: 10.1016/s0735-1097(96)00567-0.

文献AI研究员

20分钟写一篇综述，助力文献阅读效率提升50倍。

立即体验

用中文搜PubMed

大模型驱动的PubMed中文搜索引擎

马上搜索

文档翻译

学术文献翻译模型，支持多种主流文档格式。

立即体验