抗葡糖基转移酶抗体对变形链球菌体外黏附的差异性抑制作用
Differential inhibition of Streptococcus mutans in vitro adherence by anti-glucosyltransferase antibodies.
作者信息
Kuramitsu H K, Ingersoll L
出版信息
Infect Immun. 1976 Jun;13(6):1775-7. doi: 10.1128/iai.13.6.1775-1777.1976.
Abstract
Antibodies prepared against an insoluble-soluble glucan-synthesizing fraction significantly inhibited in vitro adherence of Streptococcus mutans, whereas antibodies directed against a soluble glucan-synthesizing fraction were much less inhibitory.
摘要
针对不溶性 - 可溶性葡聚糖合成组分制备的抗体能显著抑制变形链球菌的体外黏附，而针对可溶性葡聚糖合成组分的抗体抑制作用则小得多。
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本文引用的文献
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Some Immunochemical Properties of Dextransucrase and Invertase from Streptococcus mutans.变形链球菌葡聚糖蔗糖酶和转化酶的一些免疫化学性质。
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Mechanism of the Adherence of Streptococcus mutans to Smooth Surfaces III. Purification and Properties of the Enzyme Complex Responsible for Adherence.变形链球菌黏附于光滑表面的机制 III. 负责黏附的酶复合物的纯化和特性。
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Extracellular glucosyltransferase activity of an HS strain of Streptococcus mutans.变形链球菌HS菌株的细胞外葡糖基转移酶活性
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Synthesis of insoluble dextran and its significance in the formation of gelatinous deposits by plaque-forming streptococci.不溶性葡聚糖的合成及其在形成菌斑的链球菌产生凝胶状沉积物过程中的意义。
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Dental caries: prospects for prevention.龋齿：预防前景
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Demonstration of five serological groups of streptococcal strains resembling Streptococcus mutans.与变形链球菌相似的链球菌菌株的五个血清学组的证明。
Odontol Revy. 1970;21(2):143-52.
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Inhibition of glucosyltransferase activity by antisera to known serotypes of Streptococcus mutans.变形链球菌已知血清型抗血清对葡糖基转移酶活性的抑制作用。
Infect Immun. 1973 Feb;7(2):237-41. doi: 10.1128/iai.7.2.237-241.1973.
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Structural and enzymatic studies on glucans synthesized with glucosyltransferases of some strains of oral streptococci.对某些口腔链球菌菌株的葡糖基转移酶合成的葡聚糖进行的结构和酶学研究。
Acta Chem Scand. 1972;26(6):2223-30. doi: 10.3891/acta.chem.scand.26-2223.
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Adherence inhibition of Streptococcus mutans: an assay reflecting a possible role of antibody in dental caries prophylaxis.变形链球菌的黏附抑制：一种反映抗体在预防龋齿中可能作用的检测方法。
Infect Immun. 1972 Apr;5(4):419-27. doi: 10.1128/iai.5.4.419-427.1972.
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The structure of water-insoluble glucans of cariogenic Streptococcus mutans, formed in the absence and presence of dextranase.
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