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Hydrolase levels in necrotizing and non-necrotizing BCG-induced pulmonary granulomas.

作者信息

McGee M P, Myrvik Q N

出版信息

J Reticuloendothel Soc. 1976 Sep;20(3):187-95.

PMID:978658
Abstract
摘要

相似文献

1
Hydrolase levels in necrotizing and non-necrotizing BCG-induced pulmonary granulomas.坏死性和非坏死性卡介苗诱导的肺肉芽肿中的水解酶水平。
J Reticuloendothel Soc. 1976 Sep;20(3):187-95.
2
Isolation and characterization of hydrolase-containing granules from rabbit lung macrophages.
J Reticuloendothel Soc. 1974 Sep;16(3):184-92.
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Serum angiotensin-converting-enzyme in rabbits with and without pulmonary granulomatosis. Granulomatosis induced with complete-Freund's-adjuvant or BCG.患有和未患有肺肉芽肿病的兔子的血清血管紧张素转换酶。用完全弗氏佐剂或卡介苗诱导肉芽肿病。
Sarcoidosis. 1986 Mar;3(1):60-6.
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On the chemical pathology of interstitial fluid. 2. Intracellular and interstitial enzyme activity in experimental granulomas.论组织液的化学病理学。2. 实验性肉芽肿中的细胞内和组织内酶活性
Acta Pathol Microbiol Scand. 1968;72(2):205-18.
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[Change of lung lavage levels of angiotensin converting enzyme activity in mice with BCG-induced granulomatous pulmonary inflammation].[卡介苗诱导的小鼠肉芽肿性肺炎模型肺灌洗中血管紧张素转换酶活性水平的变化]
Nihon Kyobu Shikkan Gakkai Zasshi. 1984 Jun;22(6):474-8.
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Histoenzymology of the lung. II. The behaviour of lung hydrolytic enzymes during some experimental pulmonary processes.肺的组织酶学。II. 某些实验性肺部病变过程中肺水解酶的表现
Morphol Embryol (Bucur). 1977 Apr-Jun;23(2):151-7.
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Morphogenesis and pathogenesis of chronic lung diseases. XI. Histoenzymatic study of the experimentally developing lung granulomas.慢性肺部疾病的形态发生与发病机制。XI. 实验性肺肉芽肿形成过程中的组织酶学研究。
Morphol Embryol (Bucur). 1976 Jan-Mar;22(1):47-51.
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[Localization and chronologic changes of angiotensin-converting enzyme in experimental pulmonary granulomas by the substrate film technique. Comparison with serum ACE activity and tissue ACE activity].[采用底物膜技术对实验性肺肉芽肿中血管紧张素转换酶的定位及时间变化。与血清血管紧张素转换酶活性和组织血管紧张素转换酶活性的比较]
Kekkaku. 1984 Jan;59(1):27-31.
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[Chronologic changes of activities of macrophage-associated nonspecific esterases in the developing and healing experimental pulmonary granulomas].[实验性肺肉芽肿形成与愈合过程中巨噬细胞相关非特异性酯酶活性的时序变化]
Nihon Kyobu Shikkan Gakkai Zasshi. 1984 May;22(5):363-9.
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Enzymes in tuberculous lesions hydrolyzing protein, hyaluronic acid and chondroitin sulfate: a study of isolated macrophages and developing and healing rabbit BCG lesions with substrate film techniques; the shift of enzyme pH optima towards neutrality in "intact" cells and tissues.结核病变中水解蛋白质、透明质酸和硫酸软骨素的酶:利用底物膜技术对分离的巨噬细胞以及兔卡介苗病变发展和愈合过程的研究;“完整”细胞和组织中酶的最适pH向中性转变。
J Reticuloendothel Soc. 1974 Oct;16(4):220-231.

引用本文的文献

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Selective suppression of granuloma formation and delayed hypersensitivity in rabbits.兔肉芽肿形成和迟发型超敏反应的选择性抑制
Infect Immun. 1980 Apr;28(1):269-76. doi: 10.1128/iai.28.1.269-276.1980.
2
Tissue factor activity. A marker of alveolar macrophage maturation in rabbits. Effects of granulomatous pneumonitis.组织因子活性。家兔肺泡巨噬细胞成熟的标志物。肉芽肿性肺炎的影响。
J Clin Invest. 1984 Jun;73(6):1524-31. doi: 10.1172/JCI111358.
3
Cathepsin B and D activity in alveolar macrophages from rats with pulmonary granulomatous inflammation or acute lung injury.
患有肺部肉芽肿性炎症或急性肺损伤的大鼠肺泡巨噬细胞中组织蛋白酶B和D的活性
Agents Actions. 1989 Nov;28(3-4):264-71. doi: 10.1007/BF01967413.
4
Biochemical quantitation and histochemical localization of cathepsin B, dipeptidyl peptidases I and II, and acid phosphatase in pulmonary granulomatosis and fibrosis in rats.大鼠肺肉芽肿病和肺纤维化中组织蛋白酶B、二肽基肽酶I和II以及酸性磷酸酶的生化定量和组织化学定位
Inflammation. 1988 Feb;12(1):67-86. doi: 10.1007/BF00915893.
5
Phagocytosis-induced injury of normal and activated alveolar macrophages.吞噬作用诱导的正常及活化肺泡巨噬细胞损伤。
Infect Immun. 1979 Dec;26(3):910-5. doi: 10.1128/iai.26.3.910-915.1979.