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婴儿猝死综合征。一项前瞻性研究。

Sudden infant death syndrome. A prospective study.

作者信息

Naeye R L, Ladis B, Drage J S

出版信息

Am J Dis Child. 1976 Nov;130(11):1207-10. doi: 10.1001/archpedi.1976.02120120041005.

DOI:10.1001/archpedi.1976.02120120041005
PMID:984002
Abstract

One hundred twenty-five sudden infant death syndrome (SIDS) victims followed up since birth from a large prospective study were compared with matched controls. Some of the future SIDS victims showed evidences of neonatal brain dysfunction including abnormalities in respiration, feeding, temperature regulation, and specific neurologic tests. These abnormalities could not be ralated to events in labor or delivery. A greater proportion of the future victims were mildly underweight for gestational age. The gestations that produced the SIDS victims were characterized by a greater frequency of mothers who smoked cigarettes and had anemia. The demographic profile of SIDS families proved to be indentical to the profile for families with excessive perinatal mortality. Many of the SIDS victims showed a retardation in postnatal growth prior to death.

摘要

对一项大型前瞻性研究中自出生起就进行随访的125名婴儿猝死综合征(SIDS)受害者与匹配的对照组进行了比较。一些未来的SIDS受害者表现出新生儿脑功能障碍的迹象,包括呼吸、喂养、体温调节及特定神经学检查方面的异常。这些异常与分娩过程中的事件无关。未来受害者中,有更大比例的婴儿按孕周计算轻度体重不足。导致SIDS受害者的孕期特点是,母亲吸烟和患贫血的频率更高。事实证明,SIDS家庭的人口统计学特征与围产期死亡率过高的家庭特征相同。许多SIDS受害者在死亡前出现出生后生长发育迟缓。

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1
Sudden infant death syndrome. A prospective study.婴儿猝死综合征。一项前瞻性研究。
Am J Dis Child. 1976 Nov;130(11):1207-10. doi: 10.1001/archpedi.1976.02120120041005.
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Evolution and the sudden infant death syndrome (SIDS) : Part II: Why human infants?进化与婴儿猝死综合征(SIDS):第二部分:为什么是人类婴儿?
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