Musher D M, Schell R F, Knox J M
Cutis. 1976 Apr;17(4):739-42.
We believe that humoral and cellular immunity both are involved in the response of the host to infection with T. pallidum. A complex interaction between stimulation and suppression of cell-mediated immunity may be involved. This interaction may explain the unusual clinical course in which secondary syphilis waxes and wanes before culminating in immunity.
我们认为，体液免疫和细胞免疫均参与宿主对梅毒螺旋体感染的反应。细胞介导的免疫刺激与抑制之间可能存在复杂的相互作用。这种相互作用可能解释了二期梅毒在最终产生免疫力之前病情起伏不定的异常临床病程。
