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特发性脊柱侧凸的发病机制：神经与骨骼生长失衡？

The pathogenesis of idiopathic scoliosis: uncoupled neuro-osseous growth?

作者信息

Porter R W

出版信息

Eur Spine J. 2001 Dec;10(6):473-81. doi: 10.1007/s005860100311.

DOI:10.1007/s005860100311
PMID:11806387
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC3611539/
Abstract

This paper examines the following speculative hypothesis: "that in some patients with scoliosis there is disproportionate neuro-osseous growth--the longitudinal growth of the spinal cord fails to keep pace with the growth of the vertebral column and, as a consequence, the spine buckles into a scoliosis deformity". A literature review of the morphology and neurology of scoliosis does not deny the hypothesis. Several mechanisms are suggested as to why the spinal cord growth could become uncoupled from osseous growth.

摘要

本文探讨了以下推测性假说

“在一些脊柱侧弯患者中，存在神经 - 骨骼生长不均衡的情况——脊髓的纵向生长未能跟上脊柱的生长，因此，脊柱弯曲形成脊柱侧弯畸形”。对脊柱侧弯的形态学和神经学进行的文献综述并未否定这一假说。文中提出了几种关于脊髓生长为何会与骨骼生长脱节的机制。