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本文引用的文献
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Complement C5a receptors and neutrophils mediate fetal injury in the antiphospholipid syndrome.
J Clin Invest. 2003 Dec;112(11):1644-54. doi: 10.1172/JCI18817.
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Mutations in human complement regulator, membrane cofactor protein (CD46), predispose to development of familial hemolytic uremic syndrome.
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Evidence-based management of thrombosis in the antiphospholipid antibody syndrome.
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Mutations in factor H reduce binding affinity to C3b and heparin and surface attachment to endothelial cells in hemolytic uremic syndrome.
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Expression and function of C5a receptor in mouse microvascular endothelial cells.
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Role of membrane cofactor protein (CD46) in regulation of C4b and C3b deposited on cells.
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How antibodies to a ubiquitous cytoplasmic enzyme may provoke joint-specific autoimmune disease.
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Arthritis critically dependent on innate immune system players.
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Essential role of neutrophils in the initiation and progression of a murine model of rheumatoid arthritis.
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Arthritis provoked by linked T and B cell recognition of a glycolytic enzyme.
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