Studies on the pathogenesis of rheumatoid arthritis; clinical observations during and after a course of insulin and simultaneous administration of glucose and glucose metabolites to patients with rheumatoid arthritis.
作者信息
HAYDU G G
出版信息
Rheumatism. 1950 Jul;6(3):133-6.
PMID:15430877
Abstract
摘要
相似文献
1
Studies on the pathogenesis of rheumatoid arthritis; clinical observations during and after a course of insulin and simultaneous administration of glucose and glucose metabolites to patients with rheumatoid arthritis.类风湿性关节炎发病机制的研究；类风湿性关节炎患者在胰岛素治疗期间及之后同时给予葡萄糖和葡萄糖代谢物的临床观察。
Rheumatism. 1950 Jul;6(3):133-6.
2
Abnormal Glucose Metabolism in Rheumatoid Arthritis.类风湿关节炎中的葡萄糖代谢异常
Biomed Res Int. 2017;2017:9670434. doi: 10.1155/2017/9670434. Epub 2017 Apr 26.
3
Glucose-insulin tolerance test in rheumatoid arthritis; study of twenty-nine cases.类风湿关节炎的葡萄糖-胰岛素耐量试验；29例研究
J Am Med Assoc. 1955 Oct 8;159(6):561-2. doi: 10.1001/jama.1955.02960230025009a.
4
Rheumatoid arthritis modifies basal and insulin-mediated glucose uptake by human synoviocytes.类风湿性关节炎改变人滑膜细胞的基础葡萄糖摄取及胰岛素介导的葡萄糖摄取。
Cell Mol Biol. 1991;37(5):541-7.
5
Some preliminary observations on the treatment of rheumatoid arthritis with cortisone plus insulin.关于用可的松加胰岛素治疗类风湿性关节炎的一些初步观察
J Clin Endocrinol Metab. 1950 Jul;10(7):800-1. doi: 10.1210/jcem-10-7-800.
6
Insulin hypoglycemia in treatment of rheumatoid arthritis; preliminary observations of eleven patients.胰岛素低血糖疗法治疗类风湿关节炎；11例患者的初步观察
Perm Found Med Bull. 1952 Aug;10(1-4):119-28.
7
[Insulin resistance and rheumatoid arthritis].[胰岛素抵抗与类风湿关节炎]
Reumatol Clin. 2011 Mar-Apr;7(2):124-9. doi: 10.1016/j.reuma.2010.03.010. Epub 2011 Jan 14.
8
Spontaneous hypoglycaemia after insulin therapy in rheumatoid arthritis.类风湿关节炎胰岛素治疗后自发性低血糖症
Br Med J. 1951 Sep 8;2(4731):578-80. doi: 10.1136/bmj.2.4731.578.
9
10
Angiopoietin-like 4: A molecular link between insulin resistance and rheumatoid arthritis.血管生成素样蛋白4：胰岛素抵抗与类风湿关节炎之间的分子联系。
J Orthop Res. 2017 May;35(5):939-943. doi: 10.1002/jor.23507. Epub 2017 Mar 8.
引用本文的文献
1
High affinity glycosaminoglycan and autoantigen interaction explains joint specificity in a mouse model of rheumatoid arthritis.高亲和力糖胺聚糖与自身抗原的相互作用解释了类风湿性关节炎小鼠模型中的关节特异性。
J Biol Chem. 2009 Jan 23;284(4):2354-62. doi: 10.1074/jbc.M806458200. Epub 2008 Oct 22.
Zotero 插件