Martin P R, Marie G V, Nathan P R
Department of Psychology, University of Western Australia, Nedlands.
J Psychosom Res. 1992 Feb;36(2):137-48. doi: 10.1016/0022-3999(92)90022-t.
Psychophysiological investigations of tension headaches and migraines have produced inconsistent and inconclusive findings. The study reported here aimed to further explore headache mechanisms by repeating earlier research with two main variations. The first pertained to recording and analytic procedures: a different approach was taken to measuring and interpreting cephalic vascular activity. The second was in the experimental conditions used: pain levels were manipulated so that the psychophysiology of headaches could be studied under conditions of pain increasing and decreasing. The findings provided no support for the role of muscle tension as a headache mechanism. Tentative support was obtained for vascular models of headaches. Headaches were associated with distension of the superficial temporal artery between pressure pulses. The data suggested that local vasomotor activity might play a role in dilating the temporal artery but that the effect seemed driven by elevated blood pressure and heart rate.
对紧张性头痛和偏头痛的心理生理学研究结果并不一致且尚无定论。本文所报告的研究旨在通过重复早期研究并进行两个主要方面的变动，进一步探究头痛的机制。第一个变动涉及记录和分析程序：采用了不同的方法来测量和解释头部血管活动。第二个变动在于所使用的实验条件：对疼痛程度进行了控制，以便在疼痛加剧和减轻的条件下研究头痛的心理生理学。研究结果不支持肌肉紧张作为头痛机制的作用。对头痛的血管模型获得了初步支持。头痛与压力脉冲之间颞浅动脉的扩张有关。数据表明，局部血管运动活动可能在扩张颞动脉方面起作用，但这种作用似乎是由血压和心率升高驱动的。
