相似文献
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Suppression of lymphocyte response to concanavalin A by mucopolysaccharide material from Treponema pallidum-infected rabbits.梅毒感染兔的黏多糖物质对淋巴细胞对刀豆球蛋白A反应的抑制作用。
Infect Immun. 1979 Oct;26(1):64-9. doi: 10.1128/iai.26.1.64-69.1979.
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Reappraisal of lymphocyte responsiveness to concanavalin A during experimental syphilis: evidence that glycosaminoglycans in the sera and tissues interfere ith active binding sites on the lectin and not with the lymphocytes.实验性梅毒期间淋巴细胞对刀豆球蛋白A反应性的重新评估：血清和组织中的糖胺聚糖干扰凝集素上的活性结合位点而非淋巴细胞的证据。
Infect Immun. 1982 Feb;35(2):552-9. doi: 10.1128/iai.35.2.552-559.1982.
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Depression of lymphocyte response to concanavalin A in rabbits infected with Treponema pallidum (Nichols strain).感染梅毒螺旋体（Nichols株）的家兔对刀豆球蛋白A的淋巴细胞反应受抑制。
Infect Immun. 1976 Jul;14(1):320-2. doi: 10.1128/iai.14.1.320-322.1976.
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Selective in vitro response of thymus-derived lymphocytes from Treponema pallidum-infected rabbits.梅毒螺旋体感染兔胸腺来源淋巴细胞的体外选择性反应。
Infect Immun. 1977 Dec;18(3):603-11. doi: 10.1128/iai.18.3.603-611.1977.
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In vitro cell response of Treponema pallidum-infected rabbits. I. Lymphocyte transformation.梅毒螺旋体感染兔的体外细胞反应。I. 淋巴细胞转化
Clin Exp Immunol. 1977 Sep;29(3):480-6.
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Modulation of normal lymphocyte stimulation by serum from Treponema pallidum-infected rabbits.
Cell Immunol. 1980 Jul 15;53(1):29-38. doi: 10.1016/0008-8749(80)90423-2.
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Serum regulation of in vitro lymphocyte responses in early experimental syphilis.早期实验性梅毒中血清对体外淋巴细胞反应的调节作用
Infect Immun. 1982 Aug;37(2):568-78. doi: 10.1128/iai.37.2.568-578.1982.
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Effect of route of infection with Treponema pallidum. In vitro cellular response to treponemal antigens.梅毒螺旋体感染途径的影响。对梅毒螺旋体抗原的体外细胞反应。
Arch Pathol Lab Med. 1982 Sep;106(9):436-9.
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Serum of rabbits infected with Treponema pallidum (Nichols) inhibits in vitro transformation of normal rabbit lymphocytes.
Cell Immunol. 1979 Feb;42(2):363-72. doi: 10.1016/0008-8749(79)90201-6.
引用本文的文献
1
Sequential changes in susceptibility to Treponema pallidum of rabbits previously infected with Treponema paraluis-cuniculi.先前感染了兔梅毒螺旋体的兔子对梅毒螺旋体易感性的序贯变化。
Br J Vener Dis. 1981 Feb;57(1):11-4. doi: 10.1136/sti.57.1.11.
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Transfer of resistance with syphilitic immune cells: lack of correlation with mitogenic activity.梅毒免疫细胞耐药性的转移：与促有丝分裂活性缺乏相关性。
Infect Immun. 1982 Jan;35(1):187-92. doi: 10.1128/iai.35.1.187-192.1982.
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Reappraisal of lymphocyte responsiveness to concanavalin A during experimental syphilis: evidence that glycosaminoglycans in the sera and tissues interfere ith active binding sites on the lectin and not with the lymphocytes.实验性梅毒期间淋巴细胞对刀豆球蛋白A反应性的重新评估：血清和组织中的糖胺聚糖干扰凝集素上的活性结合位点而非淋巴细胞的证据。
Infect Immun. 1982 Feb;35(2):552-9. doi: 10.1128/iai.35.2.552-559.1982.
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Accelerated lesion development in experimental syphilis.实验性梅毒中病变的加速发展。
Infect Immun. 1981 Nov;34(2):478-82. doi: 10.1128/iai.34.2.478-482.1981.
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Helical conformation of Treponema pallidum (Nichols strain), Treponema paraluis-cuniculi, Treponema denticola, Borrelia turicatae, and unidentified oral spirochetes.梅毒螺旋体（Nichols株）、兔梅毒螺旋体、齿垢密螺旋体、杜氏疏螺旋体及未鉴定的口腔螺旋体的螺旋结构。
Infect Immun. 1981 May;32(2):937-40. doi: 10.1128/iai.32.2.937-940.1981.
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Serum regulation of in vitro lymphocyte responses in early experimental syphilis.早期实验性梅毒中血清对体外淋巴细胞反应的调节作用
Infect Immun. 1982 Aug;37(2):568-78. doi: 10.1128/iai.37.2.568-578.1982.
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Polyanions in syphilis: evidence that glycoproteins and macromolecules resembling glycosaminoglycans are synthesised by host tissues in response to infection with Treponema pallidum.梅毒中的多阴离子：有证据表明，宿主组织在感染梅毒螺旋体后会合成类似于糖胺聚糖的糖蛋白和大分子。
Br J Vener Dis. 1984 Apr;60(2):75-82. doi: 10.1136/sti.60.2.75.
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Induction of chondroitin sulfate proteoglycan synthesis and secretion in lymphocytes and monocytes.硫酸软骨素蛋白聚糖在淋巴细胞和单核细胞中的合成与分泌诱导。
J Cell Biol. 1983 Aug;97(2):351-8. doi: 10.1083/jcb.97.2.351.
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Detection of circulating immune complexes in the sera of rabbits with experimental syphilis: possible role in immunoregulation.实验性梅毒兔血清中循环免疫复合物的检测：在免疫调节中的可能作用。
Infect Immun. 1980 Aug;29(2):575-82. doi: 10.1128/iai.29.2.575-582.1980.
本文引用的文献
1
Hyaluronidase and experimental syphilis. III. Metachromasia in syphilitic orchitis and its relationship to hyaluronic acid.透明质酸酶与实验性梅毒。III. 梅毒性睾丸炎中的异染性及其与透明质酸的关系。
Am J Syph Gonorrhea Vener Dis. 1950 Nov;34(6):501-14.
2
Chondroitin sulfate and hyaluronic acid in syphilomas of cortisone-treated rabbits.可的松治疗兔梅毒瘤中的硫酸软骨素和透明质酸
Science. 1956 Aug 10;124(3215):275. doi: 10.1126/science.124.3215.275.
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Morphologic and histochemical sequences in syphilitic and in tuberculous orchitis in the rabbit.兔梅毒和结核性睾丸炎的形态学及组织化学变化过程
Am J Syph Gonorrhea Vener Dis. 1954 May;38(3):189-202.
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Assessment of the "luotest" in late syphillis.“罗氏试验”在晚期梅毒中的评估。
Br J Vener Dis. 1966 Jun;42(2):119-21. doi: 10.1136/sti.42.2.119.
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Runting syndrome in neonatal rabbits infected with Treponema pallidum.感染梅毒螺旋体的新生兔发育迟缓综合征
Clin Exp Immunol. 1967 May;2(3):311-20.
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In vitro lymphocyte response to Treponema refringens im human syphilis.人梅毒中对致密螺旋体的体外淋巴细胞反应。
Infect Immun. 1974 Apr;9(4):654-7. doi: 10.1128/iai.9.4.654-657.1974.
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The microassay of hyaluronic acid concentration and hyaluronidase activity by capillary turbidity (CT) and capillary turbidity reduction (CTR) tests.
Anal Biochem. 1973 Dec;56(2):428-34. doi: 10.1016/0003-2697(73)90208-x.
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Leucocyte migration and cell-mediated immunity in syphilis.梅毒中的白细胞迁移与细胞介导免疫
Br J Vener Dis. 1972 Dec;48(6):483-8. doi: 10.1136/sti.48.6.483.
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Why is the infectious stage of syphilis prolonged?梅毒的传染期为何会延长？
Br J Vener Dis. 1974 Feb;50(1):45-9. doi: 10.1136/sti.50.1.45.
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Reduced lymphocyte transformation due to a plasma factor in patients with active syphilis.活动性梅毒患者血浆因子导致淋巴细胞转化降低。
Lancet. 1969 Aug 2;2(7614):246-7. doi: 10.1016/s0140-6736(69)90010-5.
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