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How the bacterial pathogen Listeria monocytogenes mediates the switch from environmental Dr. Jekyll to pathogenic Mr. Hyde.

作者信息

Gray Michael J, Freitag Nancy E, Boor Kathryn J

机构信息

Department of Food Science, Cornell University, Ithaca, NY 14853, USA.

出版信息

Infect Immun. 2006 May;74(5):2505-12. doi: 10.1128/IAI.74.5.2505-2512.2006.

DOI:10.1128/IAI.74.5.2505-2512.2006
PMID:16622185
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1459693/
Abstract
摘要

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本文引用的文献

1
Pillars article: cellular resistance to infection. J. Exp. Med. 1962. 116: 381-406.支柱文章:细胞对感染的抵抗力。《实验医学杂志》1962年。第116卷:381 - 406页。
J Immunol. 2014 Oct 1;193(7):3185-221.
2
Sigma B contributes to Listeria monocytogenes gastrointestinal infection but not to systemic spread in the guinea pig infection model.在豚鼠感染模型中,西格玛B有助于单核细胞增生李斯特菌的胃肠道感染,但对其全身扩散没有作用。
Infect Immun. 2006 Feb;74(2):876-86. doi: 10.1128/IAI.74.2.876-886.2006.
3
SigmaB contributes to Listeria monocytogenes invasion by controlling expression of inlA and inlB.西格玛B通过控制内化素A(InlA)和内化素B(InlB)的表达促进单核细胞增生李斯特菌的侵袭。
Microbiology (Reading). 2005 Oct;151(Pt 10):3215-3222. doi: 10.1099/mic.0.28070-0.
4
Involvement of reactive oxygen intermediate in the enhanced expression of virulence-associated genes of Listeria monocytogenes inside activated macrophages.活性氧中间体参与活化巨噬细胞内单核细胞增生李斯特菌毒力相关基因的表达增强。
Microbiol Immunol. 2005;49(8):805-11. doi: 10.1111/j.1348-0421.2005.tb03661.x.
5
The 5' untranslated region-mediated enhancement of intracellular listeriolysin O production is required for Listeria monocytogenes pathogenicity.5'非翻译区介导的细胞内李斯特菌溶血素O产生增强是单核细胞增生李斯特菌致病性所必需的。
Mol Microbiol. 2005 Sep;57(5):1460-73. doi: 10.1111/j.1365-2958.2005.04780.x.
6
The Listeria monocytogenes prfAP2 promoter is regulated by sigma B in a growth phase dependent manner.单核细胞增生李斯特菌的prfAP2启动子受σB以生长阶段依赖性方式调控。
FEMS Microbiol Lett. 2005 Apr 15;245(2):329-36. doi: 10.1016/j.femsle.2005.03.025.
7
The mutation G145S in PrfA, a key virulence regulator of Listeria monocytogenes, increases DNA-binding affinity by stabilizing the HTH motif.单核细胞增生李斯特菌关键毒力调节因子PrfA中的G145S突变通过稳定HTH基序增加了DNA结合亲和力。
Mol Microbiol. 2005 Apr;56(2):433-46. doi: 10.1111/j.1365-2958.2005.04561.x.
8
Enhanced synthesis of internalin A in aro mutants of Listeria monocytogenes indicates posttranscriptional control of the inlAB mRNA.单核细胞增生李斯特菌aro突变体中内化素A的合成增强表明inlAB mRNA存在转录后调控。
J Bacteriol. 2005 Apr;187(8):2836-45. doi: 10.1128/JB.187.8.2836-2845.2005.
9
A PrfA-regulated bile exclusion system (BilE) is a novel virulence factor in Listeria monocytogenes.一种由PrfA调控的胆汁排除系统(BilE)是单核细胞增生李斯特菌中的一种新型毒力因子。
Mol Microbiol. 2005 Feb;55(4):1183-95. doi: 10.1111/j.1365-2958.2004.04454.x.
10
Contribution of three bile-associated loci, bsh, pva, and btlB, to gastrointestinal persistence and bile tolerance of Listeria monocytogenes.三个与胆汁相关的基因座bsh、pva和btlB对单增李斯特菌在胃肠道中的持久性和胆汁耐受性的作用。
Infect Immun. 2005 Feb;73(2):894-904. doi: 10.1128/IAI.73.2.894-904.2005.