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Biochemistry of dystrophic muscle. 2. Some enzyme changes in dystrophic mouse muscle.

作者信息

Pennington R J

机构信息

Department of Clinical Chemistry, King's College (University of Durham) and Royal Victoria Infirmary, Newcastle upon Tyne.

出版信息

Biochem J. 1963 Jul;88(1):64-8. doi: 10.1042/bj0880064.

DOI:10.1042/bj0880064
PMID:16749029
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1203848/
Abstract
摘要

相似文献

1
Biochemistry of dystrophic muscle. 2. Some enzyme changes in dystrophic mouse muscle.营养不良性肌肉的生物化学。2. 营养不良小鼠肌肉中的一些酶变化。
Biochem J. 1963 Jul;88(1):64-8. doi: 10.1042/bj0880064.
2
Differences in enzyme efflux from dystrophic mouse skeletal muscle and heart.营养不良小鼠骨骼肌和心脏中酶流出的差异。
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3
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Muscle Nerve. 1982 Jul-Aug;5(6):425-33. doi: 10.1002/mus.880050603.
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[Muscle regeneration in mdx mouse, and a trial of normal myoblast transfer into regenerating dystrophic muscle].[mdx小鼠的肌肉再生以及将正常成肌细胞移植到再生的营养不良肌肉中的试验]
Rinsho Shinkeigaku. 1990 Oct;30(10):1066-72.
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The effect of stretch removal on muscle weight and proteolytic enzyme activity in normal and dystrophic chicken muscles.拉伸去除对正常和营养不良鸡肌肉的肌肉重量及蛋白水解酶活性的影响。
Muscle Nerve. 1984 Jul-Aug;7(6):482-5. doi: 10.1002/mus.880070611.
6
Comparison of the protein-synthesizing machinery in the skeletal muscle of normal and dystrophic Bar Harbor mice.正常与营养不良的巴港小鼠骨骼肌中蛋白质合成机制的比较。
Biochem J. 1969 Nov;115(3):377-82. doi: 10.1042/bj1150377.
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Changes of soluble glycoproteins in dystrophic (dy/dy) mouse muscle shown by lectin binding.通过凝集素结合显示的营养不良(dy/dy)小鼠肌肉中可溶性糖蛋白的变化。
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IGF-II ameliorates the dystrophic phenotype and coordinately down-regulates programmed cell death.胰岛素样生长因子-II改善营养不良表型并协同下调程序性细胞死亡。
Cell Death Differ. 2000 Nov;7(11):1109-18. doi: 10.1038/sj.cdd.4400738.
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Decrease of myofiber branching via muscle-specific expression of the olfactory receptor mOR23 in dystrophic muscle leads to protection against mechanical stress.通过在营养不良性肌肉中肌肉特异性表达嗅觉受体mOR23来减少肌纤维分支,可对机械应力起到保护作用。
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Regeneration of dystrophic muscle following multiple injections of bupivacaine.多次注射布比卡因后营养不良性肌肉的再生
Muscle Nerve. 1988 Jun;11(6):588-96. doi: 10.1002/mus.880110611.

引用本文的文献

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Protein degradation in skin fibroblasts from patients with Duchenne muscular dystrophy.杜氏肌营养不良症患者皮肤成纤维细胞中的蛋白质降解
Biochem J. 1980 Oct 15;192(1):257-62. doi: 10.1042/bj1920257.
2
In vivo effects of protease inhibitors on chickens with hereditary muscular dystrophy.蛋白酶抑制剂对患有遗传性肌肉萎缩症鸡的体内作用。
J Clin Invest. 1981 Apr;67(4):969-74. doi: 10.1172/jci110147.
3
Successful treatment of murine muscular dystrophy with the proteinase inhibitor leupeptin.用蛋白酶抑制剂亮抑酶肽成功治疗小鼠肌肉萎缩症。
Proc Natl Acad Sci U S A. 1981 Dec;78(12):7742-4. doi: 10.1073/pnas.78.12.7742.
4
Adenosine 5 -triphosphate--creatine phosphotransferase from dystrophic mouse skeletal muscle. A genetic lesion associated with the catalytic-site thiol group.来自营养不良小鼠骨骼肌的三磷酸腺苷-肌酸磷酸转移酶。一种与催化位点巯基相关的基因损伤。
Biochem J. 1966 Sep;100(3):637-46. doi: 10.1042/bj1000637.
5
The determination of lactate dehydrogenase isoenzymes in normal human muscle and other tissues.正常人肌肉及其他组织中乳酸脱氢酶同工酶的测定
Biochem J. 1967 Nov;105(2):599-604. doi: 10.1042/bj1050599.
6
Cell-free protein synthesis in heart and skeletal muscles from polymyopathic hamsters.多肌病仓鼠心脏和骨骼肌中的无细胞蛋白质合成
Biochem J. 1973 Feb;132(2):193-201. doi: 10.1042/bj1320193.
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Scanning electron microscopy and cytochemical localization of carnitine acetyltransferase activity in normal and dystrophic muscle of mice.小鼠正常和营养不良肌肉中肉碱乙酰转移酶活性的扫描电子显微镜观察及细胞化学定位
Histochem J. 1973 Jul;5(4):335-42. doi: 10.1007/BF01004801.
8
Lysosomal activation in mouse skeletal muscle induced by protamine in vitro.
Cell Tissue Res. 1978 Jan 9;186(1):1-11. doi: 10.1007/BF00219650.
9
Protein synthesis in muscles from normal and dystrophic hamsters.正常和营养不良仓鼠肌肉中的蛋白质合成。
Biochem J. 1979 Apr 15;180(1):51-8. doi: 10.1042/bj1800051.
10
Therapeutic trials in muscular dystrophy. 1. Gold in murine dystrophy.肌肉萎缩症的治疗试验。1. 小鼠萎缩症中的金标准。
J Neurol Neurosurg Psychiatry. 1978 May;41(5):404-7. doi: 10.1136/jnnp.41.5.404.

本文引用的文献

1
Dehydrogenase Activities in Dystrophic Mice.营养不良小鼠的脱氢酶活性。
Science. 1960 Sep 2;132(3427):621-2. doi: 10.1126/science.132.3427.621.
2
Dystrophia Muscularis: A HEREDITARY PRIMARY MYOPATHY IN THE HOUSE MOUSE.肌营养不良症:家鼠中的一种遗传性原发性肌病。
Proc Natl Acad Sci U S A. 1955 Dec 15;41(12):1079-84. doi: 10.1073/pnas.41.12.1079.
3
Triphosphopyridine nucleotide (TPN) diaphorase and TPN-dependent dehydrogenase activity of reactive macrophages in tissue necrosis.组织坏死中反应性巨噬细胞的三磷酸吡啶核苷酸(TPN)黄递酶及TPN依赖性脱氢酶活性
Nature. 1962 Mar 3;193:895. doi: 10.1038/193895a0.
4
Creatine phosphorylkinase in muscles of dystrophic mice.营养不良小鼠肌肉中的肌酸磷酸激酶
Proc Soc Exp Biol Med. 1962 Mar;109:696-7. doi: 10.3181/00379727-109-27310.
5
5'-Adenylic acid deaminase in dystrophic mouse muscle.营养不良小鼠肌肉中的5'-腺苷酸脱氨酶
Nature. 1961 Dec 2;192:884-5. doi: 10.1038/192884a0.
6
Proteolytic activity of rat skeletal muscle. I. Evidence for the existence of an enzyme active optimally at pH 8.5 to 9.0.大鼠骨骼肌的蛋白水解活性。I. 存在一种在pH 8.5至9.0时活性最佳的酶的证据。
J Biol Chem. 1960 Mar;235:665-8.
7
Phosphoglucomutase activity in hereditary muscular dystrophy in mice.小鼠遗传性肌肉萎缩症中的磷酸葡萄糖变位酶活性
Proc Soc Exp Biol Med. 1959 Oct-Dec;102:720-2. doi: 10.3181/00379727-102-25375.
8
Enzyme studies in muscular dystrophy. I. Muscle proteolytic activity and vitamin E-deficiency.肌肉萎缩症中的酶研究。I. 肌肉蛋白水解活性与维生素E缺乏症。
Proc Soc Exp Biol Med. 1955 Feb;88(2):257-60. doi: 10.3181/00379727-88-21556.
9
Increased lysosomal enzymes in genetic muscular dystrophy.遗传性肌肉萎缩症中溶酶体酶增加。
Arch Biochem Biophys. 1962 Feb;96:340-6. doi: 10.1016/0003-9861(62)90418-6.
10
Biochemistry of dystrophic muscle. Mitochondrial succinate-tetrazolium reductase and adenosine triphosphatase.营养不良性肌肉的生物化学。线粒体琥珀酸 - 四氮唑还原酶和三磷酸腺苷酶。
Biochem J. 1961 Sep;80(3):649-54. doi: 10.1042/bj0800649.