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1
Interleukin-1 administration to C3H/HeJ mice after but not prior to infection increases resistance to Salmonella typhimurium.在感染后而非感染前向C3H/HeJ小鼠注射白细胞介素-1可增强其对鼠伤寒沙门氏菌的抵抗力。
Infect Immun. 1991 Dec;59(12):4729-31. doi: 10.1128/iai.59.12.4729-4731.1991.
2
Formation of interferon-gamma and tumor necrosis factor in mice during Salmonella typhimurium infection.鼠伤寒沙门氏菌感染期间小鼠体内干扰素-γ和肿瘤坏死因子的形成。
Pathobiology. 1991;59(3):194-6. doi: 10.1159/000163643.
3
Bacterial lipoprotein induces resistance to Gram-negative sepsis in TLR4-deficient mice via enhanced bacterial clearance.细菌脂蛋白通过增强细菌清除作用诱导TLR4缺陷小鼠对革兰氏阴性菌败血症产生抗性。
J Immunol. 2005 Jan 15;174(2):1020-6. doi: 10.4049/jimmunol.174.2.1020.
4
Exogenous tumor necrosis factor alpha and interleukin-1 alpha increase resistance to Salmonella typhimurium: efficacy is influenced by the Ity and Lps loci.外源性肿瘤坏死因子α和白细胞介素-1α可增强对鼠伤寒沙门氏菌的抵抗力：其疗效受Ity和Lps基因座的影响。
Infect Immun. 1995 Aug;63(8):3196-8. doi: 10.1128/iai.63.8.3196-3198.1995.
5
Treatment of mice with IL-1 before infection increases resistance to a lethal challenge with Salmonella typhimurium. The effect correlates with the resistance allele at the Ity locus.在感染前用白细胞介素-1治疗小鼠可增强其对鼠伤寒沙门氏菌致死性攻击的抵抗力。该效应与Ity基因座上的抗性等位基因相关。
J Immunol. 1994 Jul 1;153(1):212-9.
6
Toll-like receptor expression in C3H/HeN and C3H/HeJ mice during Salmonella enterica serovar Typhimurium infection.鼠伤寒沙门氏菌感染期间C3H/HeN和C3H/HeJ小鼠中Toll样受体的表达
Infect Immun. 2003 Nov;71(11):6653-7. doi: 10.1128/IAI.71.11.6653-6657.2003.
7
Evidence for separate genetic defects in C3H/HeJ and C3HeB/FeJ mice, that affect susceptibility to gram-negative infections.C3H/HeJ和C3HeB/FeJ小鼠中存在影响革兰氏阴性菌感染易感性的不同遗传缺陷的证据。
J Immunol. 1985 Jun;134(6):4118-22.
8
Induction of activated macrophages in C3H/HeJ mice by avirulent Salmonella.无毒力沙门氏菌对C3H/HeJ小鼠中活化巨噬细胞的诱导作用。
J Immunol. 1988 Mar 1;140(5):1638-44.
9
Immunity to Salmonella typhimurium infection in C3H/HeJ and C3H/HeNCrlBR mice: studies with an aromatic-dependent live S. typhimurium strain as a vaccine.C3H/HeJ和C3H/HeNCrlBR小鼠对鼠伤寒沙门氏菌感染的免疫：以一种芳香族依赖性活鼠伤寒沙门氏菌菌株作为疫苗的研究
Infect Immun. 1985 Mar;47(3):605-12. doi: 10.1128/iai.47.3.605-612.1985.
10
Macrophage defect and inflammatory cell recruitment dysfunction in Salmonella susceptible C3H/HeJ mice.
Cell Immunol. 1986 Oct 1;102(1):68-77. doi: 10.1016/0008-8749(86)90326-6.

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Role of the caspase-1 inflammasome in Salmonella typhimurium pathogenesis.胱天蛋白酶-1炎性小体在鼠伤寒沙门氏菌致病机制中的作用。
J Exp Med. 2006 Jun 12;203(6):1407-12. doi: 10.1084/jem.20060206. Epub 2006 May 22.
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Differential production of interleukin-12 mRNA by murine macrophages in response to viable or killed Salmonella spp.小鼠巨噬细胞对活的或灭活的沙门氏菌作出反应时白细胞介素-12 mRNA的差异产生
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Interleukin-1 is involved in mouse resistance to Mycobacterium avium.白细胞介素-1参与小鼠对鸟分枝杆菌的抗性。
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Antimicrob Agents Chemother. 1993 Dec;37(12):2688-92. doi: 10.1128/AAC.37.12.2688.
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Dynamics of avian inflammatory response to Salmonella-immune lymphokines. Changes in avian blood leukocyte populations.禽类对沙门氏菌免疫淋巴细胞因子的炎症反应动力学。禽类血液白细胞群体的变化。
Inflammation. 1994 Aug;18(4):373-88. doi: 10.1007/BF01534435.
6
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J Clin Invest. 1995 Aug;96(2):884-92. doi: 10.1172/JCI118135.
7
Exogenous tumor necrosis factor alpha and interleukin-1 alpha increase resistance to Salmonella typhimurium: efficacy is influenced by the Ity and Lps loci.外源性肿瘤坏死因子α和白细胞介素-1α可增强对鼠伤寒沙门氏菌的抵抗力：其疗效受Ity和Lps基因座的影响。
Infect Immun. 1995 Aug;63(8):3196-8. doi: 10.1128/iai.63.8.3196-3198.1995.

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Genetic control of responses to bacterial lipopolysaccharides in mice. I. Evidence for a single gene that influences mitogenic and immunogenic respones to lipopolysaccharides.小鼠对细菌脂多糖反应的遗传控制。I. 影响对脂多糖促有丝分裂和免疫原性反应的单个基因的证据。
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Expression, purification and characterization of recombinant murine granulocyte-macrophage colony-stimulating factor and bovine interleukin-2 from yeast.酵母来源的重组小鼠粒细胞巨噬细胞集落刺激因子和牛白细胞介素-2的表达、纯化及特性分析
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Independence of macrophage activation and expression of the alleles of the Ity (immunity to typhimurium) locus.巨噬细胞激活与Ity（鼠伤寒免疫）基因座等位基因表达的独立性。
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在感染后而非感染前向C3H/HeJ小鼠注射白细胞介素-1可增强其对鼠伤寒沙门氏菌的抵抗力。

Interleukin-1 administration to C3H/HeJ mice after but not prior to infection increases resistance to Salmonella typhimurium.

作者信息

Morrissey P J, Charrier K

机构信息

Immunex Corporation, Seattle, Washington 98101.

出版信息

Infect Immun. 1991 Dec;59(12):4729-31. doi: 10.1128/iai.59.12.4729-4731.1991.

DOI:10.1128/iai.59.12.4729-4731.1991
PMID:1937834
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC259108/
Abstract

Interleukin-1 (IL-1) treatment of C3H/HeN and C3H/HeJ mice prior to infection with Salmonella typhimurium increased the survival fraction only in C3H/HeN mice. IL-1 administration after infection resulted in a significant increase in mean survival time in C3H/HeJ but not C3H/HeN mice. Bacterial growth in IL-1-treated C3H/HeJ mice was less than that in control mice.

摘要

在用鼠伤寒沙门氏菌感染之前，对白三烯-1（IL-1）处理过的C3H/HeN和C3H/HeJ小鼠进行观察，发现只有C3H/HeN小鼠的存活比例有所增加。感染后给予IL-1，结果显示C3H/HeJ小鼠的平均存活时间显著增加，而C3H/HeN小鼠则不然。经IL-1处理的C3H/HeJ小鼠体内的细菌生长情况低于对照小鼠。