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A functional differentiation of human neutrophil granules: generation of C5a by a specific (secondary) granule product and inactivation of C5a by azurophil (primary) granule products.

作者信息

Wright D G, Gallin J I

出版信息

J Immunol. 1977 Sep;119(3):1068-76.

PMID:19531
Abstract
摘要

相似文献

1
A functional differentiation of human neutrophil granules: generation of C5a by a specific (secondary) granule product and inactivation of C5a by azurophil (primary) granule products.人类中性粒细胞颗粒的功能分化：由特定（次级）颗粒产物产生C5a以及嗜天青（初级）颗粒产物使C5a失活。
J Immunol. 1977 Sep;119(3):1068-76.
2
The sequential release of granule constitutents from human neutrophils.人中性粒细胞颗粒成分的顺序释放。
J Immunol. 1980 Feb;124(2):855-62.
3
The discharge of primary and secondary granules during immune phagocytosis by normal and chronic granulocytic leukaemia polymorphonuclear neutrophils.正常和慢性粒细胞白血病多形核中性粒细胞在免疫吞噬过程中初级和次级颗粒的释放。
Br J Haematol. 1982 Jun;51(2):201-8.
4
C5 chemotactic fragments produced by an enzyme in lysosomal granules of neutrophils.由中性粒细胞溶酶体颗粒中的一种酶产生的C5趋化片段。
J Immunol. 1970 Mar;104(3):535-43.
5
Human neutrophil N-acetyl-beta-D-glucosaminidase: granule localization. Further evidence for two azurophil granules.人中性粒细胞N-乙酰-β-D-氨基葡萄糖苷酶：颗粒定位。关于两种嗜天青颗粒的进一步证据。
J Lab Clin Med. 1984 Jul;104(1):60-8.
6
Expression of azurophil and specific granule proteins during differentiation of NB4 cells in neutrophils.NB4细胞向中性粒细胞分化过程中嗜天青颗粒蛋白和特异性颗粒蛋白的表达
J Cell Physiol. 1998 May;175(2):203-10. doi: 10.1002/(SICI)1097-4652(199805)175:2<203::AID-JCP10>3.0.CO;2-9.
7
Generation of a neutrophil chemotactic agent by spermatozoa: role of complement and regulation by seminal plasma factors.精子产生中性粒细胞趋化因子：补体的作用及精浆因子的调节
J Immunol. 1976 Oct;117(4):1378-86.
8
Neutrophil specific granule deficiency.中性粒细胞特异性颗粒缺乏症。
Annu Rev Med. 1985;36:263-74. doi: 10.1146/annurev.me.36.020185.001403.
9
Intracellular control of human neutrophil secretion. I. C5a-induced stimulus-specific desensitization and the effects of cytochalasin B.人类中性粒细胞分泌的细胞内控制。I. C5a诱导的刺激特异性脱敏作用及细胞松弛素B的影响。
J Immunol. 1978 Sep;121(3):851-5.
10
Complement-induced expression of cryptic receptors on the neutrophil surface: a mechanism for regulation of acute inflammation in trauma.补体诱导中性粒细胞表面隐蔽受体的表达：一种创伤后急性炎症调节机制
Surgery. 1984 Aug;96(2):336-44.

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Resolution of Inflammation after Skeletal Muscle Ischemia-Reperfusion Injury: A Focus on the Lipid Mediators Lipoxins, Resolvins, Protectins and Maresins.骨骼肌缺血再灌注损伤后炎症的消退：聚焦脂质介质脂氧素、消退素、保护素和促消退介素
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Chemotactic factors released in culture by intact developing and healing skin lesions produced in rabbits by the irritant sulfur mustard.
由刺激性芥子气在兔身上造成的完整的正在发展和愈合的皮肤损伤在培养物中释放的趋化因子。
Inflammation. 1997 Apr;21(2):251-67. doi: 10.1023/a:1027378422627.
4
Development of chemotactic responsiveness in myeloid precursor cells: studies with a human leukemia cell line.髓系前体细胞趋化反应性的发育：对一种人类白血病细胞系的研究。
Proc Natl Acad Sci U S A. 1980 Jun;77(6):3664-8. doi: 10.1073/pnas.77.6.3664.
5
Demonstration of a local exhaustion of complement components and of an enzymatic degradation of immunoglobulins in pleural empyema: a possible factor favouring the persistence of local bacterial infections.胸膜积脓中补体成分局部耗竭及免疫球蛋白酶解的证明：一个有利于局部细菌感染持续存在的可能因素。
Clin Exp Immunol. 1980 Dec;42(3):506-14.
6
A chemotactic inhibitor in synovial fluid.滑液中的一种趋化抑制剂。
Immunology. 1983 May;49(1):131-8.
7
Abnormal regulation of inflammatory skin responses in male patients with chronic granulomatous disease.慢性肉芽肿病男性患者炎症性皮肤反应的异常调节
Inflammation. 1983 Sep;7(3):227-32. doi: 10.1007/BF00917259.
8
Subcellular localization of the large subunit of Mo1 (Mo1 alpha; formerly gp 110), a surface glycoprotein associated with neutrophil adhesion.与中性粒细胞黏附相关的表面糖蛋白Mo1（Mo1α；原称gp 110）大亚基的亚细胞定位。
J Clin Invest. 1984 Oct;74(4):1280-90. doi: 10.1172/JCI111538.
9
Development of the adult respiratory distress syndrome: progressive alteration of neutrophil chemotactic and secretory processes.成人呼吸窘迫综合征的发展：中性粒细胞趋化和分泌过程的渐进性改变。
Am J Pathol. 1984 Sep;116(3):427-35.
10
Human neutrophil chemotactic response to group A streptococci: bacteria-mediated interference with complement-derived chemotactic factors.人类中性粒细胞对A组链球菌的趋化反应：细菌介导对补体衍生趋化因子的干扰
Infect Immun. 1983 Jan;39(1):239-46. doi: 10.1128/iai.39.1.239-246.1983.