Bustos Patricia, Muñoz Sergio, Vargas Claudio, Amigo Hugo
Departamento Nutrición, Facultad de Medicina, Universidad de Chile, Santiago, Chile.
Salud Publica Mex. 2009 May-Jun;51(3):187-93. doi: 10.1590/s0036-36342009000300008.
To estimate the effect of indigenous ancestry and poverty on nutritional outcomes in Chilean schoolchildren.
We used the national database of children entering to the public educational system in 1997-2004. This includes anthropometric assessment, socioeconomic status and parental surnames, used to derive the ethnic origin. Logistic regression models related poverty and ethnicity on stunting and obesity were done, controlling for sex, age and calendar year.
Data convey 1,580,103 children being 7.4% indigenous; 2.9% had stunting and around 16.0% were obese. Stratifying by poverty, it was shown that the poorest had higher risk of stunting both in indigenous (OR= 2.30; CI95%=2.27-2.33) and non indigenous (OR= 2.29; CI95%= 2.28-2.30). Conversely, poverty was a 'protective factor' for obesity (OR= 0.63; CI95%= 0.62-0.64). Indigenous origin showed a significant OR slightly over the null.
In Chilean children, poverty is a risk factor for stunting but still protects from obesity, independent of indigenous origin.
评估智利学龄儿童的本土血统和贫困状况对营养状况的影响。
我们使用了1997 - 2004年进入公共教育系统的儿童的全国数据库。这包括人体测量评估、社会经济地位和父母姓氏，用于确定种族出身。针对发育迟缓与肥胖，构建了逻辑回归模型，将贫困和种族作为变量，同时控制性别、年龄和年份。
数据涵盖1,580,103名儿童，其中7.4%为本土血统；2.9%发育迟缓，约16.0%肥胖。按贫困程度分层显示，最贫困群体在本土血统儿童（比值比=2.30；95%置信区间=2.27 - 2.33）和非本土血统儿童（比值比=2.29；95%置信区间=2.28 - 2.30）中发育迟缓风险均更高。相反，贫困是肥胖的“保护因素”（比值比=0.63；95%置信区间=0.62 - 0.64）。本土血统显示出略高于零假设的显著比值比。
在智利儿童中，贫困是发育迟缓的风险因素，但仍是预防肥胖的因素，且不受本土血统影响。
