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Mutators and hypermutability in bacteria: the Escherichia coli paradigm.

作者信息

Jayaraman R

机构信息

R. H. 35, Palaami Enclave, New Natham Road, Madurai 625 014, India.

出版信息

J Genet. 2009 Dec;88(3):379-91. doi: 10.1007/s12041-009-0058-2.

DOI:10.1007/s12041-009-0058-2
PMID:20086310
Abstract
摘要

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1
Mutators and hypermutability in bacteria: the Escherichia coli paradigm.细菌中的突变体与超突变性:以大肠杆菌为范例
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The DNA repair helicase UvrD is essential for replication fork reversal in replication mutants.DNA修复解旋酶UvrD对于复制突变体中的复制叉逆转至关重要。
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3
Hypermutability and error catastrophe due to defects in ribonucleotide reductase.由于核糖核苷酸还原酶的缺陷导致的高突变率和错误灾难。
Proc Natl Acad Sci U S A. 2013 Nov 12;110(46):18596-601. doi: 10.1073/pnas.1310849110. Epub 2013 Oct 28.
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The Spectrum of Replication Errors in the Absence of Error Correction Assayed Across the Whole Genome of .在没有错误校正的情况下,对. 的整个基因组进行复制错误分析。
Genetics. 2018 Aug;209(4):1043-1054. doi: 10.1534/genetics.117.300515. Epub 2018 Jun 15.
5
Overproduction of DnaE protein (alpha subunit of DNA polymerase III) restores viability in a conditionally inviable Escherichia coli strain deficient in DNA polymerase I.DnaE蛋白(DNA聚合酶III的α亚基)的过量产生可恢复在DNA聚合酶I缺陷的条件性非存活大肠杆菌菌株中的生存能力。
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Directionality of DNA replication fork movement strongly affects the generation of spontaneous mutations in Escherichia coli.DNA复制叉移动的方向性强烈影响大肠杆菌中自发突变的产生。
J Mol Biol. 2001 Apr 13;307(5):1195-206. doi: 10.1006/jmbi.2001.4557.
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Mutations produced by DNA polymerase III holoenzyme of Escherichia coli after in vitro synthesis in the absence of single-strand binding protein.在无单链结合蛋白的情况下进行体外合成后,大肠杆菌DNA聚合酶III全酶产生的突变。
Mol Microbiol. 1990 Oct;4(10):1645-52. doi: 10.1111/j.1365-2958.1990.tb00541.x.
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Mutator mutants of Escherichia coli carrying a defect in the DNA polymerase III tau subunit.携带DNA聚合酶III τ亚基缺陷的大肠杆菌诱变突变体。
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Saturation of mismatch repair in the mutD5 mutator strain of Escherichia coli.大肠杆菌mutD5突变菌株中错配修复的饱和
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Levels of epsilon, an essential replication subunit of Escherichia coli DNA polymerase III, are controlled by heat shock proteins.大肠杆菌DNA聚合酶III的必需复制亚基ε的水平受热休克蛋白调控。
J Bacteriol. 1992 Dec;174(23):7509-16. doi: 10.1128/jb.174.23.7509-7516.1992.

引用本文的文献

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A Comprehensive View of Translesion Synthesis in Escherichia coli.大肠杆菌中跨损伤合成的全面观察
Microbiol Mol Biol Rev. 2020 Jun 17;84(3). doi: 10.1128/MMBR.00002-20. Print 2020 Aug 19.
2
Genes and Proteomes Associated With Increased Mutation Frequency and Multidrug Resistance of Naturally Occurring Mismatch Repair-Deficient Hypermutators.与自然发生的错配修复缺陷型高突变体的突变频率增加和多药耐药性相关的基因和蛋白质组
Front Microbiol. 2020 May 8;11:770. doi: 10.3389/fmicb.2020.00770. eCollection 2020.
3
Resistance mechanisms.耐药机制。

本文引用的文献

1
Mutability and importance of a hypermutable cell subpopulation that produces stress-induced mutants in Escherichia coli.在大肠杆菌中产生应激诱导突变体的超突变细胞亚群的可变性及其重要性。
PLoS Genet. 2008 Oct 3;4(10):e1000208. doi: 10.1371/journal.pgen.1000208.
2
The relationship between dNTP pool levels and mutagenesis in an Escherichia coli NDP kinase mutant.大肠杆菌NDP激酶突变体中dNTP库水平与诱变之间的关系。
Proc Natl Acad Sci U S A. 2008 Jul 22;105(29):10197-202. doi: 10.1073/pnas.0802816105. Epub 2008 Jul 10.
3
Spontaneous mutagenesis associated with nucleotide excision repair in Escherichia coli.
Ann Transl Med. 2016 Sep;4(17):326. doi: 10.21037/atm.2016.09.14.
4
The evolution of bacterial mutation rates under simultaneous selection by interspecific and social parasitism.细菌在种间寄生和社会寄生的同时选择下突变率的演变。
Proc Biol Sci. 2013 Nov 6;280(1773):20131913. doi: 10.1098/rspb.2013.1913. Print 2013 Dec 22.
5
Role of DNA base excision repair in the mutability and virulence of Streptococcus mutans.DNA 碱基切除修复在变形链球菌突变和毒力中的作用。
Mol Microbiol. 2012 Jul;85(2):361-77. doi: 10.1111/j.1365-2958.2012.08116.x. Epub 2012 Jun 15.
6
Hypermutation and stress adaptation in bacteria.细菌中的超突变与应激适应
J Genet. 2011 Aug;90(2):383-91. doi: 10.1007/s12041-011-0086-6.
大肠杆菌中与核苷酸切除修复相关的自发诱变
Genes Cells. 2008 May;13(5):459-69. doi: 10.1111/j.1365-2443.2008.01185.x.
4
Role of accessory DNA polymerases in DNA replication in Escherichia coli: analysis of the dnaX36 mutator mutant.辅助DNA聚合酶在大肠杆菌DNA复制中的作用:dnaX36突变体的分析
J Bacteriol. 2008 Mar;190(5):1730-42. doi: 10.1128/JB.01463-07. Epub 2007 Dec 21.
5
Coevolution with viruses drives the evolution of bacterial mutation rates.与病毒的共同进化推动了细菌突变率的进化。
Nature. 2007 Dec 13;450(7172):1079-81. doi: 10.1038/nature06350. Epub 2007 Dec 2.
6
Mutation as a stress response and the regulation of evolvability.作为应激反应的突变与进化能力的调控。
Crit Rev Biochem Mol Biol. 2007 Sep-Oct;42(5):399-435. doi: 10.1080/10409230701648502.
7
Stress-induced mutagenesis in bacteria.细菌中的应激诱导诱变
Crit Rev Biochem Mol Biol. 2007 Sep-Oct;42(5):373-97. doi: 10.1080/10409230701648494.
8
What a difference a decade makes: insights into translesion DNA synthesis.十年间变化如此之大:对跨损伤DNA合成的见解。
Proc Natl Acad Sci U S A. 2007 Oct 2;104(40):15591-8. doi: 10.1073/pnas.0704219104. Epub 2007 Sep 26.
9
The microbiology of mutability.可变性的微生物学
FEMS Microbiol Lett. 2007 Dec;277(1):11-20. doi: 10.1111/j.1574-6968.2007.00901.x. Epub 2007 Aug 22.
10
Adaptive amplification.自适应放大
Crit Rev Biochem Mol Biol. 2007 Jul-Aug;42(4):271-83. doi: 10.1080/10409230701507757.