免疫学。类风湿关节炎中的纵火犯。
Immunology. Arsonists in rheumatoid arthritis.
机构信息
Department of Medicine, University of Utah, Salt Lake City, UT 84112, USA.
出版信息
Science. 2010 Jan 29;327(5965):528-9. doi: 10.1126/science.1185869.
Abstract
Inflammation associated with rheumatoid arthritis is stoked by platelets, expanding their roles into the immune system.
摘要
与类风湿性关节炎相关的炎症是由血小板引起的，这扩大了它们在免疫系统中的作用。
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本文引用的文献
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Platelets amplify inflammation in arthritis via collagen-dependent microparticle production.血小板通过胶原依赖性微粒生成在关节炎中放大炎症反应。
Science. 2010 Jan 29;327(5965):580-3. doi: 10.1126/science.1181928.
2
PMNs facilitate translocation of platelets across human and mouse epithelium and together alter fluid homeostasis via epithelial cell-expressed ecto-NTPDases.中性粒细胞促进血小板跨人及小鼠上皮细胞的转运，并通过上皮细胞表达的胞外核苷三磷酸二磷酸酶共同改变液体稳态。
J Clin Invest. 2008 Nov;118(11):3682-92. doi: 10.1172/JCI35874. Epub 2008 Oct 16.
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Signal-dependent protein synthesis by activated platelets: new pathways to altered phenotype and function.活化血小板的信号依赖性蛋白质合成：改变表型和功能的新途径。
Arterioscler Thromb Vasc Biol. 2008 Mar;28(3):s17-24. doi: 10.1161/ATVBAHA.107.160218.
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Platelet-mediated modulation of adaptive immunity: unique delivery of CD154 signal by platelet-derived membrane vesicles.血小板介导的适应性免疫调节：血小板衍生膜囊泡对CD154信号的独特传递
Blood. 2008 May 15;111(10):5028-36. doi: 10.1182/blood-2007-06-097410. Epub 2008 Jan 15.
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Platelet microparticles are heterogeneous and highly dependent on the activation mechanism: studies using a new digital flow cytometer.血小板微粒具有异质性，且高度依赖于激活机制：使用新型数字流式细胞仪的研究
Cytometry A. 2007 Jan;71(1):38-45. doi: 10.1002/cyto.a.20354.
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Escaping the nuclear confines: signal-dependent pre-mRNA splicing in anucleate platelets.逃离细胞核限制：无核血小板中信号依赖的前体mRNA剪接
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