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Indices of neural function in autism: clinical and biologic implications.

作者信息

Minshew N J

机构信息

Western Psychiatric Institute and Clinic, University of Pittsburgh, Pennsylvania.

出版信息

Pediatrics. 1991 May;87(5 Pt 2):774-80.

PMID:2020536
Abstract
摘要

相似文献

1
Indices of neural function in autism: clinical and biologic implications.
Pediatrics. 1991 May;87(5 Pt 2):774-80.
2
Clinical and neurophysiological aspects of epilepsy in subjects with autism and mental retardation.自闭症和智力障碍患者癫痫的临床及神经生理学方面
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3
[Electrophysiologic aspects of infantile autism].
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Auditory evoked fields predict language ability and impairment in children.听觉诱发电位可预测儿童的语言能力和语言障碍。
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Neurophysiological mechanisms in psychotic children (EEG and ERSP) studies).
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[Contribution of neurophysiology to our knowledge of autism].
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8
[Infantile autism. Functional examinations].[婴儿自闭症。功能检查]
Soins Psychiatr. 1992 Jun-Jul(140-141):17-20.
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Failure to deactivate in autism: the co-constitution of self and other.自闭症中无法去激活:自我与他人的共同构成。
Trends Cogn Sci. 2006 Oct;10(10):431-3. doi: 10.1016/j.tics.2006.08.002. Epub 2006 Aug 23.
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Autism: the first firm finding = underconnectivity?自闭症:首个确凿发现 = 连接不足?
Epilepsy Behav. 2007 Aug;11(1):20-4. doi: 10.1016/j.yebeh.2007.03.010. Epub 2007 May 24.

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Using quantitative and analytic EEG methods in the understanding of connectivity in autism spectrum disorders: a theory of mixed over- and under-connectivity.运用定量和分析脑电图方法理解自闭症谱系障碍中的连接:混合过度和连接不足的理论。
Front Hum Neurosci. 2014 Feb 26;8:45. doi: 10.3389/fnhum.2014.00045. eCollection 2014.
2
Hyperglycosylation and reduced GABA currents of mutated GABRB3 polypeptide in remitting childhood absence epilepsy.缓解期儿童失神癫痫中突变型GABRB3多肽的高糖基化和GABA电流降低
Am J Hum Genet. 2008 Jun;82(6):1249-61. doi: 10.1016/j.ajhg.2008.04.020.
3
Assessment in multisite randomized clinical trials of patients with autistic disorder: the Autism RUPP Network. Research Units on Pediatric Psychopharmacology.
孤独症谱系障碍患者多中心随机临床试验的评估:孤独症RUPP网络。儿科精神药理学研究单位。
J Autism Dev Disord. 2000 Apr;30(2):99-111. doi: 10.1023/a:1005451304303.
4
Etiology of infantile autism: a review of recent advances in genetic and neurobiological research.婴儿自闭症的病因:遗传与神经生物学研究的最新进展综述
J Psychiatry Neurosci. 1999 Mar;24(2):103-15.
5
Oculomotor evidence for neocortical systems but not cerebellar dysfunction in autism.自闭症中存在新皮质系统而非小脑功能障碍的动眼神经证据。
Neurology. 1999 Mar 23;52(5):917-22. doi: 10.1212/wnl.52.5.917.
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Brief report: electroencephalographic paroxysmal activities in the frontal area emerged in middle childhood and during adolescence in a follow-up study of autism.简短报告:在一项自闭症随访研究中,额区脑电图阵发性活动出现在童年中期和青春期。
J Autism Dev Disord. 1997 Oct;27(5):605-20. doi: 10.1023/a:1025886228387.
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Subgroups of autistic children based on social behavior display distinct patterns of brain activity.基于社交行为的自闭症儿童亚组表现出不同的大脑活动模式。
J Abnorm Child Psychol. 1995 Oct;23(5):569-83. doi: 10.1007/BF01447662.
8
Startle modulation studies in autism.
J Autism Dev Disord. 1993 Dec;23(4):619-37. doi: 10.1007/BF01046105.