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Kidney development in the absence of Gdnf and Spry1 requires Fgf10.
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Canonical Notch signaling in the developing lung is required for determination of arterial smooth muscle cells and selection of Clara versus ciliated cell fate.
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Neuropeptide Y functions as a facilitator of GDNF-induced budding of the Wolffian duct.
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The contribution of Notch1 to nephron segmentation in the developing kidney is revealed in a sensitized Notch2 background and can be augmented by reducing Mint dosage.
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Advances in early kidney specification, development and patterning.
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Etv4 and Etv5 are required downstream of GDNF and Ret for kidney branching morphogenesis.
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Inactivation of Notch signaling in the renal collecting duct causes nephrogenic diabetes insipidus in mice.
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The Id2+ distal tip lung epithelium contains individual multipotent embryonic progenitor cells.
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