Inflammation and neurogenic hypertension: a new role for the circumventricular organs?
作者信息
Lazartigues Eric
出版信息
Circ Res. 2010 Jul 23;107(2):166-7. doi: 10.1161/CIRCRESAHA.110.224873.
Abstract
摘要
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本文引用的文献
1
Central and peripheral mechanisms of T-lymphocyte activation and vascular inflammation produced by angiotensin II-induced hypertension.血管紧张素Ⅱ诱导的高血压导致 T 淋巴细胞激活和血管炎症的中枢和外周机制。
Circ Res. 2010 Jul 23;107(2):263-70. doi: 10.1161/CIRCRESAHA.110.217299. Epub 2010 Jun 17.
2
Induction of hypertension and peripheral inflammation by reduction of extracellular superoxide dismutase in the central nervous system.中枢神经系统中超氧化物歧化酶减少诱导高血压和外周炎症。
Hypertension. 2010 Feb;55(2):277-83, 6p following 283. doi: 10.1161/HYPERTENSIONAHA.109.142646. Epub 2009 Dec 14.
3
Regulation of central angiotensin type 1 receptors and sympathetic outflow in heart failure.心力衰竭时中枢血管紧张素1型受体的调节与交感神经传出活动
Am J Physiol Heart Circ Physiol. 2009 Nov;297(5):H1557-66. doi: 10.1152/ajpheart.00073.2009. Epub 2009 Aug 28.
4
Brain renin angiotensin in disease.疾病中的脑肾素血管紧张素
J Mol Med (Berl). 2008 Jun;86(6):715-22. doi: 10.1007/s00109-008-0331-5. Epub 2008 Apr 2.
5
Role of the T cell in the genesis of angiotensin II induced hypertension and vascular dysfunction.T细胞在血管紧张素II诱导的高血压和血管功能障碍发生中的作用。
J Exp Med. 2007 Oct 1;204(10):2449-60. doi: 10.1084/jem.20070657. Epub 2007 Sep 17.
6
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Brain Res Rev. 2007 Nov;56(1):119-47. doi: 10.1016/j.brainresrev.2007.06.002. Epub 2007 Jun 18.
7
Differential expression of neuronal ACE2 in transgenic mice with overexpression of the brain renin-angiotensin system.脑肾素-血管紧张素系统过表达的转基因小鼠中神经元血管紧张素转换酶2的差异表达。
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Physiology of local renin-angiotensin systems.局部肾素-血管紧张素系统的生理学
Physiol Rev. 2006 Jul;86(3):747-803. doi: 10.1152/physrev.00036.2005.
9
Contribution of the subfornical organ to angiotensin II-induced hypertension.穹窿下器在血管紧张素II诱导的高血压中的作用。
Am J Physiol Heart Circ Physiol. 2005 Feb;288(2):H680-5. doi: 10.1152/ajpheart.00823.2004. Epub 2004 Sep 30.
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Hypertension caused by angiotensin II infusion involves increased superoxide production in the central nervous system.血管紧张素 II 输注引起的高血压涉及中枢神经系统中超氧化物生成增加。
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